This quiz, titled 'Block 13 Pituitary Hypothal Disorder Drugs,' assesses knowledge on the pituitary gland's function, embryology, and hormonal interactions. It covers topics such as hormone production by somatotrophs, the origin of the neurohypophysis, and hormone receptor interactions, essential for medical students and professionals.
Aldosterone
Vasopressin
Oxytocin
Cortisol
Epinephrine
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Conivaptan
Sermorelin
Desmopressin
Octreotide
Bromocriptine
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Luteunizing Hormone (LH)
Thyroid Stimulating Hormone (TSH)
Follicle Stimulating Hormone FSH)
Oxytocin
Prolactin (Prl)
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The anterior pituitary is derived from the mesencephalon
Rathke’s pouch is derived from the posterior pituitary
The Pars Distalis is derived from endoderm
Mesoderm gives rise to the anterior pituitary
Neuroectoderm gives rise to the neurohypophysis
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The patient’s short stature is most likely explained by inadequate hypothalamic release of GHRH
The patients’ short stature is most likely explained by inability of somatotrophs to release GH.
The liver cannot produce insulin-like growth factor-1 (IGF-1).
Somatotrophes of the anterior pituitary are inactive because of hypersecretion of somatostatin.
Endogenous GHRH had suppressed the release of somatostatin.
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TSH is derived from the hypothalamus
TSH is stored in the pars nervosa of the pituitary gland
Herring bodies store oxytocin and FSH
Somatotrophs are acidophils and they produce GH
Chromophobes secrete oxytocin and vasopressin
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Increased CRH, increased ACTH, and increased cortisol
Decreased CRH, decreased ACTH, and decreased cortisol
Increased CRH, decreased ACTH, and decreased cortisol
Increased CRH, increased ACTH, and decreased cortisol
Decreased CRH, decreased ACTH, and increased cortisol
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Activating IP3-DAG-Ca+2 pathway
Activating cAMP pathway
Activating cGMP-NO pathway
Activating Na+-K+ channels
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Somatotropin
Conivaptan
Bromocriptine
Octreotide
Cabergoline
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A
B
C
D
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Hypoglycemia
Decreased gluconeogenesis
Insulin resistance
Decreased protein synthesis
Increased storage of fat in adipose tissue
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The increased venous return to the heart
The decreased water diuresis
The cerebral vasodilation
The positive inotropic effect
The decreased cardiac automaticity
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Somatrem
Sermorelin
Somatropin
Octreotide
Cabergoline
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Chemical signals are only exposed to specific cells
Some cells are completely without receptors
Some cells are completely without ligands
Signal chemicals often break down before reaching the target.
Some cells lack the necessary receptors
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Somatropin
Pegvisomant
Octreotide
Desmopressin
Cabergoline
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ACTH is derived from the hypothalamus
Herring bodies store oxytocin and vasopressin
TSH is stored in the pars nervosa of the pituitary gland
Acidophils produce FSH and ACTH
Chromophils secrete oxytocin and vasopressin
Aquatrophs of the anterior pituitary are not being stimulated by vasopressin releasing hormone.
V1 receptors on epithelial cells of the collecting tubules and ducts are absent.
V2 receptors on epithelial cells of the collecting tubules and ducts are absent.
High plasma concentration of vasopressin acting at magnocellular neurons of the paraventricular nucleus of the hypothalamus has suppressed hypothalamic release of vasopressin.
Magnocellular neurons of the paraventricular nucleus of the hypothalamus are not responding to an increase in whole body osmolarity and are not releasing vasopressin from the posterior pituitary
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Blocking prolactin receptors
Blocking receptors for prolactin releasing hormone
A cytotoxic effect on pituitary adenoma cells
Activating receptors for prolactin-inhibiting hormone
Stimulating the breakdown of prolactin
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Disappearance of cortisol producing adrenal cortical cells
Nonfunctional corticotrophs of the anterior pituitary.
Nonfunctional corticotrophs of the posterior pituitary.
Insufficient production of CRF by hypothalamic neurons.
Absence of ACTH receptors on adrenal cortical cells.
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Sermorelin
Conivaptan
Pegvisomant
Octreotide
Somatrem
Desmopressin
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