Block 7 problem quest tests knowledge in medical scenarios, including endocrinology, neonatology, and immunology. It assesses understanding of disease mechanisms, diagnosis, and genetic implications in clinical settings, essential for medical professionals.
Prolonged stimulation of the endometrium by estrogen
Prolonged stimulation of the endometrium by progesterone
Prolonged stimulation of the endometrium by estrogen and progesterone
Lack of stimulation of the endometrium
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Testosterone
Progesterone
Estradiol
Fibronectin
Serine proteases
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The granulosa layer of cells of a secondary follicle is highly vascularized
The theca externa produces androstenedione
The basal lamina separates the primary oocyte from the granulosa layer
The zona pellucida found in the secondary follicle is composed of GAGs and glycoproteins
The secondary follicle contains a secondary oocyte
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Gonadotropin releasing hormone receptor
Luteinizing hormone receptor
Follicle stimulating hormone receptor
Estrogen receptor
Progesterone receptor
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Blastocyst invasion
Formation of the placenta
Spiral artery remodeling
Luteal‐placental shift
Syncytiotrophoblast hCG production
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Stimulates vasodilation and uterine blood flow
Stimulates endometrial decidualization
Stimulates the secretion of luteal progesterone
Stimulates vascular remodeling of the spiral arteries
Stimulates blastocyst invasion
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Testesterone and dehydrotestosterone
Dehydrotestosterone and prolactin
Testosterone and follicle stimulating hormone
Luteinizing hormone and follicle stimulating hormone
Testosterone and luteinizing hormone
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The ras protooncogene is mutated at one nucleotide so that it is less able to hydrolyze bound GTP
The 3’ end of the c-src gene is deleted so that the SRC protein is always activated
There is a translocation of the c-myc gene to one of the immunoglobulin loci
There is a deletion of the p16 gene which allows unregulated passage through the G1 restriction point
. EBV causes the Her-2 protein to dimerize without a ligand binding to the receptor
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1/50
1/100
1/200
1/300
1/400
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Measurement error
Nonadditive genes
Nonshared environment
Assortative mating
Shared environment
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Replacement of the LH surge by an FSH surge.
Abolition of the LH surge
Enhanced positive feedback of the hypothalamic-pituitary-gonadal axis.
Increased conversion of testosterone to estradiol.
Inadequate decidualization of the uterus.
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GLC secrete estrogen and progesterone; TLC secrete androgen and estrogen
GLC secrete progesterone and estrogen; TLC secrete androgen and progesterone
GLC secrete androgen and estrogen; TLC secrete progesterone and estrogen
GLC secrete progesterone and androgen; TLC secrete progesterone and androgen
GLC secrete progesterone and androgen; TLC secrete androgen and estrogen
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Progesterone
Estradiol
Human chorionic gonadotropin
Estriol
Estrone
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A CGG expansion that resulted in the disruption of the promoter of the dystrophin gene
Infidelity
A point mutation in the dystrophin gene
A recombination event in the dystrophin gene that gave rise to a frameshift mutation leading to an untranslatable mRNA
A translocation that resulted in the disruption of the dystrophin gene
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Decreasing mRNA synthesis
Increasing mRNA synthesis
Creating splicing defect
Gain of function mutation
Dominant negative mutation
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Glutamic acid
Testosterone
Gamma-aminobutyric acid
Neuropeptide Y
Melatonin
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Neonatal jaundice
Dubin-Johnson syndrome
Rotor (-Manahan-Florentin) syndrome
Uridyl glucuronyl transferase deficiency
Glucose-6-phosphate dehydrogenase deficiency
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Hypoxanthine guanine phosphoribosyl transferase (HGPRT)
Purine nucleoside phosphorylase
Adenine phosphoribosyl transferase (APRT)
Adenosine desaminase (ADA)
Adenosine kinase
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Ovarian artery
Uterine artery
Cervical artery
Vesical artery
Umbilical artery
Obturator internus muscle - pudendal nerve
Levator ani muscle - pudendal nerve
Coccygeus muscle - pudendal nerve
Levator ani muscle - perineal nerve
Coccygeus muscle - perineal nerve
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Absence of GnRH surge
Prolonged elevation of follicle stimulating hormone
Increase in plasma leptin
Absence of a surge in luteinizing hormone
Abnormally elevated plasma estradiol
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FSH stimulates the primordial follicles to develop
LH stimulates proliferation of the granulosa layer of cells in the multilamina primary follicle
Activin stimulates proliferation of the follicular layer to form the granulosa layer in the multilaminar primary follicle
FSH inhibits the production of progesterone in the corpus luteum
LH inhibits the production of estrogen in the corpus luteum
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External spermatic fascia & dartos fascia
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