.
Tachypnea, tachycardia & confusion
PO2 & Exposure Time
PaO2, SaO2 & SpO2
Suspected Carbon Monoxide poisoning
PO2 & exposure time
COPD patients with chronic hypercapnia
The lungs & CNS
Excessive blood O2 levels cause retinal vasoconstriction & necrosis
Oxygen Toxicity
Fire (not spontaneous combustion)
Worsened Pneumothorax (Don't use if diagnosed)
Ear or sinus trauma (ie busted eardrum)
50%
28%
35%
80%
Oxygen toxicity
Retinopathy of prematurity
Absorption atelectasis
Oxygen induced hypoventilation
Increased PaO2 in babies
CPAP or IS
Substernal pain
Respiratory distress syndrome
PaO2 < 80
FIO2 < 60
Hypoxemia that doesn't respond to O2
All of the above
Cough
Substernal Pain,
Lethargy, Dyspnea
Refractory Hypoxemia, Vomiting
Induced Hypoventilation
Oxygen Toxicity
Induced Hyperventilation
All of the above
Prolonged exposure to high FiO2
Tachypnea, tachycardia & confusion
O2 enriched environments & surgical suites in presence of hyperbaric O2 therapy.
COPD patients with chronic hypercapnia
Prolonged exposure to high FiO2
Excessive blood O2 levels cause retinal vasoconstriction & necrosis
Patients breathing small tidal volumes with FiO2 above 0.50 are at great risk
All of the above
ROP-retinopathy of prematurity
Decrease in PaO2, an increase in CO2
Hypoxic drive
All of the above
Hypoxic Drive
Decreased PaO2
Increased PaCO2
All of the above
Decrease in PaO2, an increase in CO2
Hypoxic Drive
Retinopathy of prematurity
All of the above
Correct documented or suspected acute Hypoxemia
Decrease symptoms associated with chronic hypoxemia
Prolonged exposure to high FiO2
Decrease workload hypoxemia imposes on cardiopulmonary system
The lungs & CNS
PaO2, SaO2 & SpO2
PO2 & exposure time
All of the above
To treat or prevent hypoxemia
Decrease myocardial workload
Assessment for the need of FIO2
All of the above
Assess the patient an determine if O2 treatment is still required
Lower oxygen in the blood
Increase O2 Blood Saturation
Increase anything below 40 mm Hg
undergoes initial assessment
Evaluated for protocol criteria
Receives a treatment plan that is modified according to need
Stops receiving therapy as soon as it is no longer needed
Purpose
Patient
Hospital
Performance
Aerosol Mask
Nasal Prongs
Non-Rebreather Mask
Simple Mask
Option 1
Option 2
Option 3
Option 4
Patients breathing small tidal volumes with FiO2 above 0.50 are at great risk
O2 enriched environments
Prolonged exposure to high FiO2
Surgical suites in presence of hyperbaric O2 therapy
Hazards/complications of oxygen devices
Patients breathing small tidal volumes with FiO2 above 0.50 are at great risk
High flow nasal canula, Cascade high flow, Passover high flow, Venturie Mask, regular high flow with Aerosol mask
All of the above
Things needed in a hospital setting
None of the above
All of the above
Ways to eliminate/reduce hazards
Oxygen supports combustion, needs ignition source
Oxygen is flammable
Oxygen is highly combustable
All of the above
When patient is gagging
Bronchoscopy or long-term infant therapy
When its difficult to insert Aspiration
All of the above
Patients in O2 enriched environments & surgical suites in presence of hyperbaric O2 therapy.
Patients breathing small tidal volumes with FiO2 above 0.50 are at great risk
Patients with prolonged exposure to high FiO2
All of the above
Absorption atelectasis
Increased myocardial workload
Decreased WOB
All of the above
Option 1
Option 2
Option 3
Is greatest in patients with absorption atelectasis
Ways to reduce Absorption Atelectasis
Option 2
Option 3
Option 4
Oxygen Induced Hyperventilation
Oxygen Toxicity
Absorption Atelectasis
Oxygen Induced Hypoventilation
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