Medical Gas Therapy

1. Monitor the FIO2 closely. If greater than 0.50 it presents risk for absorption atelectasis because it washes out nitrogen and nitrogen is 80% of alveolar gas. Try not to have a patient breathing at low tidal volumes from sedation, surgical pain, of cns dysfunction...

Ways to reduce Absorption Atelectasis

Option 2

Option 3

Option 4

2. What syndrome can result from oxygen toxicity

Increased PaO2 in babies

CPAP or IS

Substernal pain

Respiratory distress syndrome

3. What are three goals of O2 Therapy

Correct documented or suspected acute Hypoxemia

Decrease symptoms associated with chronic hypoxemia

Prolonged exposure to high FiO2

Decrease workload hypoxemia imposes on cardiopulmonary system

Submit

4. Low tidal volumes from sedation,surgical pain or CNS dysfunction

Option 1

Option 2

Option 3

Is greatest in patients with absorption atelectasis

5. Check connections, Check water level, Check bags on PRB and NRB, Check flow meter or neb, Check mask for vomit, Check Pt SpO2, WOB, RR

Things needed in a hospital setting

None of the above

All of the above

Ways to eliminate/reduce hazards

6. What does O2 toxicity primarily affect?

PO2 & exposure time

COPD patients with chronic hypercapnia

The lungs & CNS

Excessive blood O2 levels cause retinal vasoconstriction & necrosis

7. The primary reason some patients with COPD hypoventilate when given O2 is most likely suppression of what

Hypoxic Drive

Decreased PaO2

Increased PaCO2

All of the above

8. List the safety rules that must be enforced during the use of oxygen therapy

Undergoes initial assessment

Evaluated for protocol criteria

Receives a treatment plan that is modified according to need

Stops receiving therapy as soon as it is no longer needed

Submit

9. High FiO2 in an infant can result in

ROP-retinopathy of prematurity

Decrease in PaO2, an increase in CO2

Hypoxic drive

All of the above

10. Who does depression of ventilation occur in?

Prolonged exposure to high FiO2

Tachypnea, tachycardia & confusion

O2 enriched environments & surgical suites in presence of hyperbaric O2 therapy.

COPD patients with chronic hypercapnia

11. What is Laboratory documentation for assessing the need for O2 Therapy?

The lungs & CNS

PaO2, SaO2 & SpO2

PO2 & exposure time

All of the above

12. What are complications/precautions associated with oxygen therapy

Oxygen toxicity

Retinopathy of prematurity

Absorption atelectasis

Oxygen induced hypoventilation

Submit

13. Same as simple mask. (bad: potential suffocation hazard). Good: Emergencies, short term, therapy requiring high FiO2. Trauma PT, Shock PT, COPD Pt

Aerosol Mask

Nasal Prongs

Non-Rebreather Mask

Simple Mask

14. How can the risk of oxygen toxicity be reduced

PaO2 < 80

FIO2 < 60

Hypoxemia that doesn't respond to O2

All of the above

15. What are determining factors for O2 toxicity?

Tachypnea, tachycardia & confusion

PO2 & Exposure Time

PaO2, SaO2 & SpO2

Suspected Carbon Monoxide poisoning

16. Fire, Dehydration, Aspiration, Pt. Becoming disconnected, Pressure ridges/reddness/sores

Hazards/complications of oxygen devices

Patients breathing small tidal volumes with FiO2 above 0.50 are at great risk

High flow nasal canula, Cascade high flow, Passover high flow, Venturie Mask, regular high flow with Aerosol mask

All of the above

17. What happens in retinopathy of prematurity?

Prolonged exposure to high FiO2

Excessive blood O2 levels cause retinal vasoconstriction & necrosis

Patients breathing small tidal volumes with FiO2 above 0.50 are at great risk

All of the above

18. If a patients SaO2 is at 90% or lower you would

Assess the patient an determine if O2 treatment is still required

Lower oxygen in the blood

Increase O2 Blood Saturation

Increase anything below 40 mm Hg

19. Nitrogen wash out which increases space between alveoli causes

Absorption atelectasis

Increased myocardial workload

Decreased WOB

All of the above

20. O2 levels above what precentage can cause Oxygen Toxicity

50%

28%

35%

80%

21. List the assessment findings that determined the choice of oxygen devices

Purpose

Patient

Hospital

Performance

Submit

22. Which one do you like?

Option 1

Option 2

Option 3

Option 4

23. List the characteristics of oxygen that make it a fire hazard

Oxygen supports combustion, needs ignition source

Oxygen is flammable

Oxygen is highly combustable

All of the above

24. When would you use a nasal catheter

When patient is gagging

Bronchoscopy or long-term infant therapy

When its difficult to insert Aspiration

All of the above

25. ABG, SpO2, level of consciousness, tachycardia, tachypnea, cyanosis

To treat or prevent hypoxemia

Decrease myocardial workload

Assessment for the need of FIO2

All of the above

26. What are clinical signs of oxygen toxicity

Cough

Substernal Pain,

Lethargy, Dyspnea

Refractory Hypoxemia, Vomiting

Submit

27. A series of reversible pathophysiologic inflammatory changes of lung tissue that can produce a progressive and lethal form of lung injury similar to acute respiratory distress syndrome-ARDS

Induced Hypoventilation

Oxygen Toxicity

Induced Hyperventilation

All of the above

28. Who is at risk of Absorption atelectasis?

Patients in O2 enriched environments & surgical suites in presence of hyperbaric O2 therapy.

Patients breathing small tidal volumes with FiO2 above 0.50 are at great risk

Patients with prolonged exposure to high FiO2

All of the above

29. What are drawbacks of HBO therapy?

Oxygen Toxicity

Fire (not spontaneous combustion)

Worsened Pneumothorax (Don't use if diagnosed)

Ear or sinus trauma (ie busted eardrum)

Submit

30. Where are O2 related fire hazards at high risk?

Patients breathing small tidal volumes with FiO2 above 0.50 are at great risk

O2 enriched environments

Prolonged exposure to high FiO2

Surgical suites in presence of hyperbaric O2 therapy

Submit

31. Complications/precautions associated with oxygen therapy

Decrease in PaO2, an increase in CO2

Hypoxic Drive

Retinopathy of prematurity

All of the above

32. 100% oxygen may result in

Oxygen Induced Hyperventilation

Oxygen Toxicity

Absorption Atelectasis

Oxygen Induced Hypoventilation

Submit

Medical Gas Therapy

1. Monitor the FIO2 closely. If greater than 0.50 it presents risk for absorption atelectasis because it washes out nitrogen and nitrogen is 80% of alveolar gas. Try not to have a patient breathing at low tidal volumes from sedation, surgical pain, of cns dysfunction...

2.

What first name or nickname would you like us to use?

2. What syndrome can result from oxygen toxicity

3. What are three goals of O2 Therapy

4. Low tidal volumes from sedation,surgical pain or CNS dysfunction

5. Check connections, Check water level, Check bags on PRB and NRB, Check flow meter or neb, Check mask for vomit, Check Pt SpO2, WOB, RR

6. What does O2 toxicity primarily affect?

7. The primary reason some patients with COPD hypoventilate when given O2 is most likely suppression of what

8. List the safety rules that must be enforced during the use of oxygen therapy

9. High FiO2 in an infant can result in

10. Who does depression of ventilation occur in?

11. What is Laboratory documentation for assessing the need for O2 Therapy?

12. What are complications/precautions associated with oxygen therapy

13. Same as simple mask. (bad: potential suffocation hazard). Good: Emergencies, short term, therapy requiring high FiO2. Trauma PT, Shock PT, COPD Pt

14. How can the risk of oxygen toxicity be reduced

15. What are determining factors for O2 toxicity?

16. Fire, Dehydration, Aspiration, Pt. Becoming disconnected, Pressure ridges/reddness/sores

17. What happens in retinopathy of prematurity?

18. If a patients SaO2 is at 90% or lower you would

19. Nitrogen wash out which increases space between alveoli causes

20. O2 levels above what precentage can cause Oxygen Toxicity

21. List the assessment findings that determined the choice of oxygen devices

22. Which one do you like?

23. List the characteristics of oxygen that make it a fire hazard

24. When would you use a nasal catheter

25. ABG, SpO2, level of consciousness, tachycardia, tachypnea, cyanosis

26. What are clinical signs of oxygen toxicity

27. A series of reversible pathophysiologic inflammatory changes of lung tissue that can produce a progressive and lethal form of lung injury similar to acute respiratory distress syndrome-ARDS

28. Who is at risk of Absorption atelectasis?

29. What are drawbacks of HBO therapy?

30. Where are O2 related fire hazards at high risk?

31. Complications/precautions associated with oxygen therapy

32. 100% oxygen may result in