This pharmacology quiz for semester 2 of 2018-2019 assesses knowledge on autonomic nervous system, cholinergic and adrenergic transmission, and actions of noradrenaline. It is designed to test key concepts such as receptor types, physiological effects, and biochemical pathways.
The vesicular ACh-H+ antiporter is a target for vesamicol.
Botulinum toxin inhibits the fusion of vesicles to presynaptic membrane.
Nicotinic receptors belong to the G-protein-coupled receptor superfamily.
Acetylcholine in the synaptic cleft is degraded by choline acetyltransferase.
Hemicholinium inhibits the synthesis of acetylcholine in the nerve terminal.
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Parasympathetic preganglionic fibres are shorter than sympathetic preganglionic fibres.
The receptors at postganglionic neuron at the sweat gland are muscarinic.
All preganglionic autonomic neurons secrete acetylcholine.
The receptors at all parasympathetic postganglionic neurons are nicotinic.
Adrenal medulla is innervated by the parasympathetic nervous system.
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Ciliary muscle of the eye.
Vagus nerve of the heart.
Sphincter muscle of the iris.
Renin-secreting cells of the kidney.
The ejaculatory muscle of the genital organ.
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It binds to receptors located on autonomic ganglia.
Binding to α2-receptors will inhibit noradrenaline release from the nerve ending.
Its action is mainly terminated by Uptake 1 mechanism.
Reserpine may depletes its storage in the vesicles.
Binding to β2-receptors causes vasoconstriction.
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An essential amino acid involved in the biosynthesis of catecholamines is tyrosine.
Noradrenaline is synthesized by DOPA-decarboxylase in the synaptic vesicle.
Cocaine interferes with the reuptake of noradrenaline into the nerve terminal.
Noradrenaline in the cytosol is degraded by catechol-O-methyl transferase
Disulfiram inhibits dopamine-β-hydroxylase leading to increased brain dopamine levels.
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