Have you been reading up on Panavelil? If so, before you do this test you must have handled test one and revised everything that was problematic on your side. Below is some review of material for test 2 in pharmacology. Do you feel confident enough to try it? Give it a shot and get to refresh your memory. Best of luck!
- rol
- one
- tropium
-tidine
Salmeterol
Cromolyn
Albuterol
Prednisolone
Albuterol
Epinephrine
Terbutaline
Salmeterol
Albuterol
Fexofenadine
Morphine
Hydrocortisone
Hydrocortisone
Epinephrine
Albuterol
Ipratropium
Treat pt with another highly specific beta 2 agonist
Increase the dosage of the medication
Switch pt to corticosteroids for 5 days
Switch pt to theophylline
-rol
-ine
-roid
-one
For maintenance and prophylaxis of asthma
For treatment of acute asthma attacks
For the treatment of candidiasis
For treatment of acute exacerbations of reversible airway obstruction in pts not responsive to first line therapies
Highly selective beta-2 agonistic effects
Inhibit synthesis of leukotrienes, prostaglandins and thromboxane
Inhibition of phosphodiesterase and antogonism of adenosine to increase cAMP.
Binds to IgE which prevents IgE from binding to anything else which would trigger allergic response
Ipratropium
Theophylline
Tiotropium
Montelukast
Omalizumab
Diphenhydramine
Ipratropium
Theophylline
Albuterol
Ipratroprium
Cromolyn
Theophylline
Beta-2 agonist
Inhibit synthesis of leukotrienes, prostaglandins and thromboxane
Phosphodiesterase inhibition and adenosine antagonism which which leads to increased cAMP.
Inhibit mast cell degranulation and prevents release of Histamine.
Does not inhibit CYP2C9, CYP3A4, or P450
Increases liver enzymes much more
Useful for acute asthma attacks
It's the only med I can actually remember.
Blockade of prostaglandins and suppression of peritumoral inflammation
Monoclonal Anti-Body which lowers IgE levels and prevnts an allergic response
Competitively blocks H1 receptors
Phosphodiesterase inhibition and adenosine antagonism which which leads to increased cAMP.
Histamine Agonists
The second generation of H1 antihistamines are HIGHLY lipid soluble
They all cross blood brain barrier and cause sedation
Known to cause HTN
-moa
-tyzine
-role
- ine
Less lipid soluble and thus cause less sedation.
Have different suffix to drug names
More likely to cause arrythmias
Are not as effective for treating allergies.
Treatment of nausea and vomiting in pregnany
Treatment of gastric acid reflux
Treatment of allergies
Prevention of motion sickness
Promethazine
Azelastine
Diphenhydramine
Fexofenadine
Desloratadine
Azelastine
Promethazine
Panavelitidine
Azelastine
Promethazine
Toxitine
Cetirizine
Cetirizine
Cyproheptadine (Periactin)
MAO inhibitor
Switch them to a different SSRI
Loratadine (Claritin)
Dimenhydrinate (Dramamine)
Cetirizine (Zyrtec)
Any of them, they have all been approved for children over the age of 6 months
With a 7 day course of Prilosec & Vancomycin
5 day of metronidazole 2x per day follwed by 10 days of Prilosec.
Place the pt on a nexium regimen.
With a 14 day course of Ranitidine, Amoxicillin, & bismuth sulfate.
- tidine
-one
-cid
-xetine
Diphenhydramine
Famotidine
Promethazine
Lansoprazole
Promotes cytochrome p450
Constipation
Gynecomastia & Galactorrhea
All the above
Ranitidine
Omeprazole
Simethicone
Misoprostol
H2 receptor blockers
H1 antagonists
Proton Pump Inhibitors
SSRI's
Simethicone
Mg(Oh)2
Sucralfate
Pantoprazole
Mg (Oh)2
Al (Oh)3
Combination of A & B
CaCO3
Gave everybody the hiccups
High risk of arrythmias
High risk of stroke
Causes erectile dysfunction and anorgasmy.
Dexamethasone
Dronabinol
Metaclopromide
Erythromycin
Dexamethasone
Promethazine
Metaclopromide
Diphenhydramine
Act on vestibular efferents within the brain stem.
Dopamine D2 antagonism at the chemoreceptor trigger zone.
Agonistic effect on CB1 type cannboid receptors on neurons around vomiting center
Blockade of prostaglandins and suppressions of peritumoral inflammation.
Diphenhydramine
Metaclopromide
Lorazepam
Dronabinol
Acts as a D2 dopamine antagonist at chemoreceptor trigger zone
An Opioid formulated with atropine and used in the treatment diarrhea.
Stimulate mu receptors to relieve pain.
A cannabinoid which acts by stimulations of CB1 type cannabinoid receptors on neurons around vomitting center.
Psyllium
Lactulose
Loperamide
Bisacodyl
In order to specifically treat inflammatory diarrhea
To reduce abuse of the agent
So that it will NOT penetrate the CNS
Just to see the reactions on the pts face when THEY figure it out.
Anticholinergic effects
Gallstone formation
CNS depression & paralytic ileus
Dependence & N/V
Increase motor activity and pepsin output
Dopamine D2 antagonism at the chemoreceptor trigger zone
Ummm... all I know is you stop pooping so much.
Inhibit acetyl choline release by activating pre-synaptic opioid receptors.
Mu, Delta, Kappa
Pi, Kappa, Alpha
Phi, Mu, Beta
Delta, Gamma, Xi
Endogenous Peptides that act on opioid receptors
A drug derived from from alkaloids of the opion poppy
Receptors of natural high that treat pain
A drug that activates some or all opiate receptor subtypes
Endogenous Peptides that act on opioid receptors
A drug derived from from alkaloids of the opium poppy
Receptors of natural high that treat pain
A drug that activates some or all opiate receptor subtypes
Endorphin
Enkalphalin
Dynorphin
Epinorphin
6-8 hours
2-4 hours
4-6 hours
1-2 hours
4-6 hours
6-8 hours
2-4 hours
1-2 hours
-tadine
-one
-phine
-lol
They are opiate receptor antagonists
Activate only the kappa receptors in order to produce analgesia
They make you feel Goooooooooood.
They are opiate receptor agonists
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