This quiz, titled 'Block 11 pulmonary Path Dr Bellot prt 1', assesses knowledge in pulmonary pathology. It covers conditions like silicosis, adenocarcinoma, and complications from smoking, providing valuable insights for medical students and professionals.
Bronchopneumonia
Bronchial asthma
Centriacinar emphysema
Bronchiectasis – kartagener syndrome
Tuberculosis
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Hypersensitivity pneumonitis
Bronchiectasis
Pleural mesothelioma
Bronchial asthma
Panacinar emphysema
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Postoperative atelectasis
Adult respiratory distress syndrome (ARDS)
Pulmonary embolism
Massive pleural effusion
Nosocomial pneumonia
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Hypersensitivity pneumonitis- farmers lung
A restrictive lung disease –coal workers pneumocosis w/ massive pulmonary fibrosis; upper>lower
An obstructive lung disease
Tuberculosis
Viral pneumonia
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Pneumococcal pneumonia
Hypersensitivity pneumonia
Secondary tuberculosis
Pancoast tumor
Sarcoidosis
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Squamous cell carcinoma
Asbestosis
Silicosis
Sarcoidosis
Undifferentiated small cell carcinoma
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There is a time lag of 20 40 years between exposure and evidence of neoplasm
All asbestos fibers are equally toxic- serpentine and amphimobile(worse one)
The biological effect of exposure is determined entirely by the weight of the fiber
All "ferruginous" bodies represent asbestos fibers
Patients need both tobacco exposure and asbestos exposure to develop mesothelioma
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Sarcoidosis
Silicosis
Asbestosis
Bagassosis
Tuberculosis
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Asbestosis
Bronchopneumonia-brocilo
Lobar pneumonia-inta alveolar
Fungal infection
Viral infection
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Large cell undifferentiated carcinoma
Adenocarcinoma
Bronchioloalveolar carcinoma
Squamous cell carcinoma
Mesothelioma
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Undifferentiated small cell carcinoma
Squamous cell carcinoma
Mesothelioma – cancer of serous membrane; easily metasticized; you are done for
Adenocarcinoma
Carcinoid tumor
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Increased pulmonary vascular resistance
Red hepatization-The first stage of hepatization of lung tissue in pneumonia, in which the exudate is blood-stained
Gray hepatization- The second stage of hepatization of lung tissue in pneumonia, when the yellowish-gray exudate is beginning to degenerate before breaking down
Subpleural adenocarcinoma
Lung abscess
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Thromboembolism
Pneumothorax
Hemothorax
Aspiration of vomitus
Massive pleural transudate
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Bronchopulmonary sequestration
Lobar pneumonia
Primary tuberculosis- combination of peripheral nodule and mediastinal lymph nodes is ghon complex
Primary atypical pneumonia
Bronchioloalveolar carcinoma
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The history of heavy smoking is unrelated to the disease
It is more commonly seen in patients younger than 40 years of age- 1°pulmonary hypertension/corpulmonale
Fibrosis of the lung is the underlying pathological problem
Enzymatic destruction of lung tissue is likely
The lung disease is usually self-healing
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Bronchial asthma
Hemorrhagic infarct of the lung
Primary pulmonary tuberculosis
Bronchopneumonia
Foreign body in the right lower lobe bronchus – obstructive/resorption atelectasis
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Bronchopneumonia –acute inflammatory infiltrates extending from bronchioles to alveoli
Lobar pneumonia-intra alveolar exudates; congestion, red hepatization;gray hepatization; resolution; most often caused by strep pneu
Viral pneumonia/interstitial pneumonia
Bronchial asthma
Bronchiectasis
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Nothing, the disease has a self-limited course and will regress spontaneously
Performing acid-fast and silver stains on the biopsy
Treat the patient with antifungal drugs
Treat the patient with anti-tuberculous drugs
Treat the patient with steroids and immunosuppressive drugs
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