This quiz, titled 'Pharm Adrenocortigoids and antag part 1', tests knowledge on corticosteroids and their antagonists, focusing on their specific functions and interactions. It assesses understanding of drugs like Dexamethasone, Spironolactone, and Mifepristone, crucial for medical and pharmacological education.
Fludrocortisone
Azathioprine
Norethindrone
Prednisone
Calcitriol
Ethinyl estradiol
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Increased catabolism of prostaglandins
Decreased number of circulating neutrophils
Increased proliferation of T cells
Increased activation of complement system
Inhibition of lymphocyte-mediated production of interleukin-2
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G-protein coupled receptors
Tyrosine kinase receptors
Specific nucleotide sequences of the DNA
Ion channel coupled receptors
Specific nucleotide sequences of RNA
Specific nucleotide sequences of ribosomes
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Autoimmune diseases
Malignancies
Infectious diseases
Inflammatory bowel diseases
Skin diseases
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Aminoglutethimide
Aldosterone
Desoxycorticosterone
Dexamethasone
Fludrocortisone
Hydrocortisone
Ketoconazole
Mifepristone
Prednisone
Spironolactone
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Aminoglutethimide
Aldosterone
Desoxycorticosterone
Dexamethasone
Fludrocortisone
Hydrocortisone
Ketoconazole
Mifepristone
Prednisone
Spironolactone
Rate this question:
Aminoglutethimide
Aldosterone
Desoxycorticosterone
Dexamethasone
Fludrocortisone
Hydrocortisone
Ketoconazole
Mifepristone
Prednisone
Spironolactone
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Cortisol induced hyperglycemia
Fludrocortisone excess
Inadequate therapy of adrenal insufficiency
Cortisol induced myopathy
Essential hypertension
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Aminoglutethimide
Aldosterone
Desoxycorticosterone
Dexamethasone
Fludrocortisone
Hydrocortisone
Ketoconazole
Mifepristone
Prednisone
Spironolactone
Rate this question:
Aminoglutethimide
Aldosterone
Desoxycorticosterone
Dexamethasone
Fludrocortisone
Hydrocortisone
Ketoconazole
Mifepristone
Prednisone
Spironolactone
Rate this question:
Aminoglutethimide
Aldosterone
Desoxycorticosterone
Dexamethasone
Fludrocortisone
Hydrocortisone
Ketoconazole
Mifepristone
Prednisone
Spironolactone
Rate this question:
Inhibition of ACTH release
Increased catabolism of prostaglandins
Increased capillary permeability
Increased release of interleukin-1
Induction of lipocortin synthesis
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Oral bioavailability is generally very good
Substantial absorption does occur through the skin
They distribute into peripheral tissues but cross the blood-brain barrier with difficulty
Their biological half-lives can be long (up to 72 hours)
They are mainly excreted unchanged by the kidney
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Decreased gluconeogenesis in liver
Increased glucose uptake by skeletal muscle
Increased glycogen synthesis in liver
Decreased glucose uptake by adipose tissue
Increased glucose utilization in peripheral tissues
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Quiz Review Timeline (Updated): Mar 20, 2023 +
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