First Patho Quiz
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Intro from Cell Adaptations to Hemodynamic disorders
- 1.What is Serous Inflammation characterized by?
- A.
The mildest form of inflammation, characterized by exudation of fluid (mostly albumin), a clear- like serum. (i.e. early stages of pneumonia)
- B.
Severe inflammation, characterized by fibrinous exudate, occurs in larger vascular tissue, seen in bacterial infections (pericarditis)
- C.
Inflammation caused by pus-forming bacteria, such as Staph or Strep. Pus may accumulate on mucous, skin, or internal organs (bacterial meningitis)
- D.
Inflammation of body surfaces of mucosa and hollow organs (stomach or intestines) or a loss of epithelial lining
- E.
Inflammation characterized by exudate of fibrin, pus, cell debris, and mucous form a pseudomembrane on the surface of ulcers
- 2.Ulcerative inflammation?
- A.
Inflammation characterized by exudate of fibrin, pus, cell debris, and mucous form a pseudomembrane on the surface of ulcers
- B.
Inflammation caused by pus-forming bacteria, such as Staph or Strep. Pus may accumulate on mucous, skin, or internal organs (bacterial meningitis)
- C.
Inflammation of body surfaces of mucosa and hollow organs (stomach or intestines) or a loss of epithelial lining
- D.
Severe inflammation, characterized by fibrinous exudate, occurs in larger vascular tissue, seen in bacterial infections (pericarditis)
- E.
The mildest form of inflammation, characterized by exudation of fluid (mostly albumin), a clear- like serum. (i.e. early stages of pneumonia)
- 3.Purulent Inflammation?
- A.
Inflammation of body surfaces of mucosa and hollow organs (stomach or intestines) or a loss of epithelial lining
- B.
The mildest form of inflammation, characterized by exudation of fluid (mostly albumin), a clear- like serum. (i.e. early stages of pneumonia)
- C.
Inflammation characterized by exudate of fibrin, pus, cell debris, and mucous form a pseudomembrane on the surface of ulcers
- D.
Inflammation caused by pus-forming bacteria (dead or dying PMNs), such as Staph or Strep. Pus may accumulate on mucous, skin, or internal organs (bacterial meningitis)
- E.
Severe inflammation, characterized by fibrinous exudate, occurs in larger vascular tissue, seen in bacterial infections (pericarditis)
- 4.Pseudomembranous Infammation?
- A.
Inflammation caused by pus-forming bacteria (dead or dying PMNs), such as Staph or Strep. Pus may accumulate on mucous, skin, or internal organs (bacterial meningitis)
- B.
Severe inflammation, characterized by fibrinous exudate, occurs in larger vascular tissue, seen in bacterial infections (pericarditis)
- C.
Inflammation characterized by exudate of fibrin, pus, cell debris, and mucous form a pseudomembrane on the surface of ulcers
- D.
Inflammation of body surfaces of mucosa and hollow organs (stomach or intestines) or a loss of epithelial lining
- E.
The mildest form of inflammation, characterized by exudation of fluid (mostly albumin), a clear- like serum. (i.e. early stages of pneumonia)
- 5.Fibrinous Inflammation?
- A.
Inflammation of body surfaces of mucosa and hollow organs (stomach or intestines) or a loss of epithelial lining
- B.
Severe inflammation, characterized by fibrinous exudate, occurs in larger vascular tissue, seen in bacterial infections (pericarditis)
- C.
Inflammation characterized by exudate of fibrin, pus, cell debris, and mucous form a pseudomembrane on the surface of ulcers
- D.
The mildest form of inflammation, characterized by exudation of fluid (mostly albumin), a clear- like serum. (i.e. early stages of pneumonia)
- E.
Inflammation caused by pus-forming bacteria (dead or dying PMNs), such as Staph or Strep. Pus may accumulate on mucous, skin, or internal organs (bacterial meningitis)
- 6.Chronic Inflammation is marked by what exudate contents?
- A.
Basophils, Neutrophils, Heparin
- B.
PMNs, fibrin, granulocytes
- C.
Fibrin, lymph, parenchymal cells
- D.
Neutrophils, macrophages, plasmin, fibrin
- E.
Lymph, macrophages, plasma cells
- 7.Granulomatous Inflammation is caused by what?
- A.
Antigens that evoke a cell mediated hypersensitivity or antigens at the site
- B.
Viral induced inflammation
- C.
A secondary liquefaction of tissue due to an initial inflammation
- D.
Dead or dying PMNs
- E.
Prolonged inflammation and irritation to tissues
- 8.The prototype for a Granulomatous disease is what?
- A.
Serous pleuritis
- B.
Ulceritive colitis
- C.
Tuberculosis
- D.
Acute bacterial meningitis
- E.
Pelvic inflammatory disease
- 9.What is an a liquid emboli?
- A.
A bolus of fat that has entered veins (usually after fx)
- B.
A large piece of tissue that become necrotic, been released and enteres vascular system
- C.
A piece of plaque from an atherosclerotic artery
- D.
Bone marrow
- E.
Air injected into veins
- 10.What is a thrombus?
- A.
A liquid bolus carried through the vascular system
- B.
Plaque that has been released from a vessel wall and floating through the vascular system
- C.
A mass of fat moving in the arterial system
- D.
A mass that was once liquid, but now solid containing blood cells and fat
- E.
Bone marrow aggregate that has entered the vascular system
- 11.Thrombi are classified as either Red or White, which type of thrombi is: smaller, composed of tightly intermixed RBC's and fibrin?
- 12.A venous emboli that reaches arterial circulation through the foramen ovale is called
- A.
Arterial
- B.
Paradoxical
- C.
Intramural
- D.
Venous
- E.
Microvascular
- 13.Arterial Emboli usually end up where?
- A.
Pulmonary arteries
- B.
Subpleaural space
- C.
Aorta
- D.
Middle cerebral artery
- E.
Left ventricle
- 14.Saddle emboli is characterized by
- A.
Large venous clots at the entry of a main pulmonary artery causing acute right heart failure and severe hypotension
- B.
A small arterial clot which will lodge in the middle cerebral artery
- C.
An aggregate of bone marrow released due to CPR
- D.
Amniotic fluid which has reached the venous system
- E.
An air embolism traveling through a vein
- 15.Insufficiences of blood supply with sudden onset, resulting in ischemic necrosis are called
- A.
Infection
- B.
Hemorrhage
- C.
Gangrene
- D.
Inflammation
- E.
Infarction
- 16.White Infarts are characterized by what type of occlusion, place of occlusion, and another prominent identifiable mark?
- 17.The only cardiac infarct that ruptures is?
- A.
Red infarct
- B.
Transmural
- C.
Paradoxical
- D.
White
- E.
Saddle emboli
- 18.Hypovolemic Shock alone results from?
- A.
Pump failure of heart
- B.
Bacterium containing LPS that die in body and cause DIC
- C.
Hypoperfusion
- D.
Loss of circulatory fluid volume
- E.
Don't know?!?
- 19.Cardiogenic shock is due to pump failure of the heart, commonly secondary to infarct. This can result in what (hint only special to the loss of pump failure)?
- A.
Waterhouse-friderechsen syndrome
- B.
Hypoperfusion
- C.
Arrythmia
- D.
Somnolence
- E.
Oligouria
- 20.Metaplasia is
- A.
Disordered growth of cells
- B.
Adaptive change from one cell type to another
- C.
Uncontrolled undifferenciation of cells
- D.
New growth of cells
- E.
Loss of functionality of cells
- 21.Hallmarks of anaplasia are:marked nuclear/cellular __________high numbers of ________________nuclear:cytoplasm ratio of _______USE COMMAS
- 22.The best example of Dysplasia is? (according to Fisch)
- A.
Squamous cell carcinoma
- B.
Melanoma
- C.
Leukemia
- D.
Dysplastic Anemia
- E.
Cervical Intraepithelial Neoplasia
- 23.Do all neoplastic cells lead to tumors?
- 24.The major difference macroscopically of benign tumors and malignant tumors is
- A.
Evenly distributed nuclei
- B.
Well developed cytoplasm
- C.
Encapsulation
- D.
New features to cells not inherent to type of cell
- E.
Uniform population
- 25.Not sharply demarcated from normal tissue, lacking sharp borders, poorly developed cytoplasm, and large nuclei are characteristics of?
- A.
Blood disorders
- B.
Macroscopic findings of melignant tumors
- C.
Microscopic findings of benign tumors
- D.
Microscopic findings of melignant tumors
- E.
Alzheimer's
- 26.An example of a lymphatic pathway of metastasis is?
- A.
Melanoma
- B.
Squamous cell carcinoma in lungs
- C.
Seeding of renal cell carcinoma to adrenal gland
- D.
Leukemia
- E.
Breast cancer
- 27.A neoplasm made up of all 3 germ layers is called?
- 28.When environmental changes exceed the ability of a cell to maintian homeostasis, adapt and cause cell death is?
- A.
Necrosis
- B.
Irreversible cell injury
- C.
Hemmorhage
- D.
Reversible cell injury
- E.
Emboli
- 29.Irreversible cell damage causes damage to the nucleus; when fragmentation of the nucleus occurs and the chromatin is distributed irregularly it is called?
- A.
Karyolysis
- B.
Karyorrhexis
- C.
Pyknosis
- D.
No bueno!
- E.
Bummer
- 30.A specialized form of liquefaction necrosis is?
- A.
Caseous Necrosis
- B.
Coagulative Necrosis
- C.
Fat Necrosis
- D.
Liquefactive Necrosis
- E.
Purulent
- 31.Autolysis is different than necrosis because it only occurs ??
- 32.Caseous Necrosis is characterized by?
- A.
Denaturing of proteins, cytoplasm appears slightly granulated
- B.
Tissue becomes soft-gel-like due to certain enzyme
- C.
Lipolytic enzymes cause liquifaction
- D.
A thick, yellow, cheesy substance forms
- E.
Necrosis
- 33.The most common form of necrosis is?
- A.
Fat
- B.
Liquifaction
- C.
Caseous
- D.
Coagulative
- E.
Gross! :-x
- 34.Necrotic tissue attracts calicium, which can undergo calcification--ranging from sand-like to rock-hard material is called?
- A.
Emboli
- B.
Metastatic Calcification
- C.
Granuloma
- D.
Dystrophic Calcification
- E.
Blah blah
- 35.A non-specific but predictable response of living tissue due to injury is called?
- 36.
![What type of Necrosis? - ProProfs]( "What type of Necrosis?")
What type of Necrosis?- A.
Caseous
- B.
Coagulative
- C.
Fat
- D.
Liquefactive
- 37.
![What type of necrosis? - ProProfs]( "What type of necrosis?")
What type of necrosis?- A.
Coagulative
- B.
Liquefactive
- C.
Caseous
- D.
Fat
- 38.
![Which type of necrosis? - ProProfs]( "Which type of necrosis?")
Which type of necrosis?- A.
Fat
- B.
Caseous
- C.
Coagulative
- D.
Liquefactive
- 39.
![Which type of necrosis is this? - ProProfs]( "Which type of necrosis is this?")
Which type of necrosis is this?- A.
Caseous
- B.
Coagulative
- C.
Fat
- D.
Liquefactive
- 40.
![What type of necrosis? - ProProfs]( "What type of necrosis?")
What type of necrosis?- A.
Caseous
- B.
Coagulative
- C.
Fat
- D.
Liquefactive
- 41.
![What type of tumor is this?? - ProProfs]( "What type of tumor is this??")
What type of tumor is this??- A.
Adenoma
- B.
Osteoma
- C.
Leiomyoma
- D.
Meningioma
- 42.
![Is this a benign tumor or melignant? - ProProfs]( "Is this a benign tumor or melignant?")
Is this a benign tumor or melignant? - 43.
![ReWhat type of necrosis is this? - ProProfs]( "ReWhat type of necrosis is this?")
ReWhat type of necrosis is this?- A.
Caseous
- B.
Coagulative
- C.
Liquefactive
- D.
Fat
- 44.
![This cell changes is one of the hallmarks of anaplasia, which one is it? - ProProfs]( "This cell changes is one of the hallmarks of anaplasia, which one is it?")
This cell changes is one of the hallmarks of anaplasia, which one is it?- A.
High mitotoc figures
- B.
Nuclei:Cytoplasm ratio
- C.
Marked pleomorhphism
- D.
Metastasis
- E.
Neoplasia
- 45.Which is not a cause of cell injury?
- A.
Ischemia
- B.
Physical trauma
- C.
Genetic Defects
- D.
Bacterial Disease
- E.
Chemical Agents
- 46.What occurs in stage 2 of cellular death?
- A.
Necrosis
- B.
Swelling of cell and release of lysosomal enzymes
- C.
Liquifying of tissues
- D.
Loss of function
- E.
Go back and look
- 47.An abnormal passageway between two endothelium-lined organs or between two blood vessels that don't normally connect
- A.
Pustule
- B.
Abscess
- C.
Anestemosis
- D.
Fistula
- 48.Has to do with anaplasia and is classified using roman numerals
- A.
Staging
- B.
Grading
- C.
TMN system
- 49.
![This is... - ProProfs]( "This is...")
This is...- A.
Caseating
- B.
Non-Caseating
- 50.
![- ProProfs]()
- A.
Caseating
- B.
Non-Caseating
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