This mid-term study quiz for Chapter 4 in Physiological Psychology covers key concepts such as drug absorption, pharmacokinetics, and the effects of dosage on the body. It assesses understanding of how drugs interact with the human body, essential for students in psychology and pharmacology fields.
Pharmacokinetics
Pharmacodynamics
Psychopharmacology
Psychopharmodynamics
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Intravenous
Intramuscular
Oral administration
Inhalation
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Care must be given to prescribing because the margin of safety is small.
The drug is incredibly safe because the median lethal dose is relatively low
The drug is incredibly safe because the median lethal dose is relatively high
The drug is not very effective
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Inactivation
Depot binding
Covalent binding
Ionic binding
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A higher dose
A lower dose
A same size dose as it would with a low affinity
The drug to be administered intravenously
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Pharmacokinetics
Pharmacodynamics
Therapeutic Index
Drug distribution
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Kidney
Liver
Stomach
Pancreas
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You can attach chemicals in a more dense form onto a lipid-soluble drug than a water-soluble drug
The immune system would recognize water-soluble drugs as invaders and fight them
The blood brain barrier is only a barrier for water-soluble molecules so lipid soluble drugs pass through
Water-soluble drugs would alter the pH value of the bloodstream and therefore have significant side effects compared to lipid-soluble drugs.
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Having to down 6 Smirnoff Twists to get buzzed when 2 used to be enough
Feeling the opposite of drunk and strongly desiring more alcohol
Feeling really sick after just one shot of tequila (and it just so happens you got really drunk and threw up after having tequila last week-end)
Feeling drunk even though your friend slipped you O'Doul's (non-alcoholic beer) as a prank
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Having to down 6 Smirnoff Twists to get buzzed when 2 used to be enough
Feeling the opposite of drunk and strongly desiring more alcohol
Feeling really sick after just one shot of tequila (and it just so happens you got really drunk and threw up after having tequila last week-end)
Feeling drunk even though your friend slipped you O'Doul's (non-alcoholic beer) as a prank
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Having to down 6 Smirnoff Twists to get buzzed when 2 used to be enough
Feeling the opposite of drunk and strongly desiring more alcohol
Feeling really sick after just one shot of tequila (and it just so happens you got really drunk and threw up after having tequila last week-end)
Feeling drunk even though your friend slipped you O'Doul's (non-alcoholic beer) as a prank
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Having to down 6 Smirnoff Twists to get buzzed when 2 used to be enough
Feeling the opposite of drunk and strongly desiring more alcohol
Feeling really sick after just one shot of tequila (and it just so happens you got really drunk and threw up after having tequila last week-end)
Feeling drunk even though your friend slipped you O'Doul's (non-alcoholic beer) as a prank
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Direct agonist
Direct antagonist
Indirect antagonist
Receptor blocker
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Direct agonist
Direct antagonist
Indirect antagonist
Drug mimic
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Direct agonist
Direct antagonist
Indirect antagonist
Drug mimic
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The ability to depot bind
The ability to activate enzymatic deactivation
Low specificity
High specificity
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They do not require neurotransmitters to activate; they activate automatically.
They are activated by neurotransmitters within the cell
They react to hormones, not neurotransmitters
They activate AChE to destroy ACh
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The body down-regulating
The body up-regulating
The body attaining lower specificity
The body attaining higher affinity
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GABA
Seretonin
Acetylcholine
Glutamate
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GABA
Seretonin
Acetylcholine
Glutamate
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M-system
Norepineprhine
A neuromodulator
D-system
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GABA
Seretonin
Acetylcholine
Glutamate
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Facilitory effects
Inhibitory effects
Activation of autoreceptors
Activation of heteroreceptors
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Synthesize more ACh
Serve as a binding receptor to ACh
Destroy ACh during enzymatic deacitvation
Serve as a catecholamine
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Metabotropic antagonist; metabotropic agonists
Metabotropic agonist; ionotropic agonists
Ionotropic antagonist; metabotropic antagonist
Ionotropic agonists; metabotropic agonist
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Dopamine
Seretonin
Epinephrine
Norepinephrine
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Tyrosine -> 5HTP -> Serotonin -> Norepinephrine
Tryptophan -> 5HTP -> Epinephrine -> Norepinephrine
Tryptophan -> L-DOPA -> Dopamine -> Norepinephrine
Tyrosine -> L-DOPA -> Dopamine -> Norepinephrine
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Dopamine
Seretonin
Epinephrine
Norepinephrine
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Dopamine
Seretonin
Epinephrine
Norepinephrine
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Tryptophan -> L-DOPA -> Serotonin
Tryptophan -> 5HTP -> Serotonin
Tyrosine -> 5HTP -> Serotonin
Tyrosine -> L-DOPA -> Serotonin
The brain
The heart
The bloodstream
The gut
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D System; dorsal raphe nucleus
V System; ventral raphe nucleus
M System; median raphe nucleus
C System; caudal raphe nucleus
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Dopamine
Seretonin
Epinephrine
Norepinephrine
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It is the principal inhibitory neurotransmitter in the brain
Tetanus toxin is a GABA antagonist by inhibiting its release
Parkinson's Disease involves degeneration of GABA cells in basal ganglia
Strychnine is a GABA antagonist by blocking its receptors
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GABA
Glycine
Glutamate
Choline
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Dopamine
GABA
Glutamate
Choline
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Reuptake cycle
Enzymatic deactivation
AChE destroys the peptides
Nothing
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Analgesia
Sedation
Distorted perception of the passage of time
Vivid visual hallucinations
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Natural analgesic; liver
Peptide neurotransmitter; synaptic vesicles
Natural ligand for THC receptor; not stored at all
Predecessor to formation of epinephrine; formed from enzymatic deactivation of norepinephrine
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Barbiturates
Dopamine receptor blockers
Ethyl Alcohol
Benzodiazepines
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Nicotine
Amphetamine
Methylenedioxymethamphetamine (MDMA)
Cocaine
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PCP
THC
Mescaline
Opiates
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Adenosine agonist
Inhibitors of serotonin or norepinephrine re-uptake
Lithium carbonate
Monoamine oxidase inhibitors
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Opiates
Antidepressant drugs
Aspirin
Opium
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GABA; decrease
GABA; increase
Glutamate; decrease
Glutamate; increase
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Reducing anxiety but suppressing dreaming (bad sleep medication)
Reducing anxiety and facilitating sleep
Making the individual happier but suppressing dreaming (bad sleep medication)
Making the individual happier and facilitating sleep
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