This quiz titled 'Block 9 Cell injury response cause necrosis MCQs' assesses knowledge on cellular responses to injury, focusing on necrosis. It features clinical scenarios related to necrosis in various tissues, testing understanding of pathophysiological processes and diagnostic skills relevant for medical students and professionals.
Caseous necrosis
Coagulative necrosis
Fibrinoid necrosis
Gangrenous necrosis
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Swelling of the mitochondria
Disruption of protein synthesis
Heterophagy by Iysosomes
Damage to the cell membrane
Karryorhexis of the nucleus
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A defect in physiologic apoptosis and developmental involution
A defect in pathologic apoptosis and developmental involution
A defect in physiologic apoptosis and organogenesis
A defect in pathologic apoptosis and cell death induced by cytotoxic T cells
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Traumatic fat necrosis
Enzymatic fat necrosis
Caseous necrosis
Coagulative necrosis and calcifications
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Barrett's esophagitis, which is an irreversible metaplastic adaptive response
Barrett's esophagitis which is an irreversible dysplastic adaptive response
Barrett's esophagitis which is a reversi ble hyperplastic adaptive response
Barrett's esophagitis which is a reversible metaplastic adaptive response
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Influx of Na+ and Ca++ resulting in liquefactive necrosis
Influx of K+ and Ca++ resulting in coagulative necrosis
Efflux of K+ and Ca++ resulting in liquefactive necrosis
Efflux of Na+ and Ca++ resulting in coagulative necrosis
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Mitochondrial swelling associated with reversible injury.
Nuclear lysis associated with irreversible injury.
Increase in intracellular calcium associated with irreversible injury.
Damage of cell membranes associated with irreversible injury.
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Protein
Lipid
Cholesterol
Pigment
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Acute purulent pneumonitis
Bronchogenic carcinoma
Granulomatous pneumonitis
Parasitic migration tracts
Pulmonary abscesses
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Abscess
Benign neoplasm
Cellulitis
Granuloma
Infarct
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Fatty change
Hydropic change
Glycogen deposition
Karyolysis
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Coagulative necrosis of myocardium
Increased serum creatinine kinase myocardiaI band (CK-MB) fraction
Left ventricle hypertrophy
Neutrophils infiltrating the myocardium
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Preservation of cardiac tissue structure without nuclei
Rupture of cardiac cell membranes
Cardiomyocyte swelling
Amorphous appearance of cardiac tissue
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Physiologic apoptosis and developmental involution.
Pathologic apoptosis and developmental involution
Physiologic apoptosis and hormonal dependent involution
Pathologic apoptosis and hormonal dependent involution
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