Questions from the pre-lab quiz, post-lab quiz, and possibly more from the section.
Hypothyroidism
Goiter development
Decreased levels of T4 secretion
Increased levels of TRH secretion
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Secreted by the thyrotropin gland, located in the neck.
Controlled by T4 secreted from the hypophysis.
Also known as triiodothyromine.
The most important hormone for maintaining the metabolic rate and body temperature.
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The stimulator of T4 production in the pituitary gland.
Produced in the pituitary gland.
Held at a low plasma concentration by low levels of TSH.
Secreted by the hypothalamus.
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The hypothalamus primarily secrets tropic hormones that stimulate the secretion of other hormones.
When plasma levels of thyroxine are low, the hypothalamus will secrete TSH and stimulate the pituitary gland to secrete more TRH.
TRH travels from the hypothalamus to the thyroid gland via the hypothalamic-thyroid portal system.
Both TRH and TSH are tropical fruit drinks.
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Catabolism refers to chemical reactions that break down large, complex molecules into smaller molecules.
Energy released in metabolism can be in the form of heat.
All of the energy from metabolism is ultimately stored in the chemical bonds of ATP.
Anabolism refers to chemical reactions that build larger, more complex molecules.
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Provide the animal T4 supplements.
Provide the animal with TSH supplements.
Provide the animal iodine and tyrosine supplements.
Provide the animal with TRH supplements.
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High body temperature
Decreased basal metabolic rate
Hyperactivity
Unexplained weight loss
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Hyperthyroidism
Development of a goiter
High levels of TRH secretion
High levels of TSH secretion
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In all cases, negative feedback mechanisms were impaired.
In all cases, TSH levels were not elevated by the thyroxine injection.
In all cases, TRH levels were increased.
In all cases, the thyroid gland response to TRH was lost.
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The TSH receptors on the thyroid gland were excessively stimulated.
The TSH receptors in the hypothalamus were being overstimulated.
The amount of T4 secretion by the thyroid gland became excessive.
The TSH receptors in the hypothalamus were overstimulated.
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hyperthyroidism
Excessive amounts of T4 secretion
Decreased levels of TSH secretion
Goiter development
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The injection decreased the negative feedback mechanism on TSH.
The amount of T4 secretion by the thyroid gland became excessive.
The secretion of TSH was inhibited by the drug.
The secretion of TRH was impaired by the drug.
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