Is autoimmune with T-Cell involvement
CNS axons are destroyed
A symptom is a shock like sensation when the patient flexes their neck (Lhermitte's sign)
Grey matter in the CNS is selectively destroyed, but not in the PNS.
Multiple sclerosis
Parkinson disease
Huntington disease
Guillain-Barre syndrome
True
False
Is associated with destruction of dopaminergic neurons
Shows Beta-amyloid plaques, Lewis bodies, and neurofibrillary tangles
Will initially show loss of long term memory
Can be treated by blocking the action of Cholinesterase
Can be treated by activating the NMDA receptor
It is a hypokinetic disorder
Dopamine deficiency is believed to be the main cause
Lewis bodies are often present in the diagnosis
A decrease in acetylcholine is present
A COMT agonist would be a poor treatment option
There is an absence of dystrophin
The defective gene is the PIG-A gene on the X chromosome
There is a decreased production of dystrophin
It is caused by an increase in acetylcholine receptor destruction at the NMJ
Myasthenia Gravis
Parkinson's disease
Muscular dystrophy
CIPD
Myasthenia Gravis
Duchenne Muscular Dystrophy
Guillan-Barre Syndrome
It will temporarily increase the amount of acetylcholine available and show temporary decreased clinical symptoms
It will temporarily activate the AchE enzyme and temporarily decrease the clinical symptoms of the disease
Atrocytoma
Meningioma
Pituitary adenoma
Craniopharyngioma
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