Mini 1 part 2 Cell Signaling quiz assesses knowledge on mechanisms like cAMP, IP3, and their roles in cellular processes. It covers topics such as hormone effects, gene transcription, and receptor differences, essential for students and professionals in biological sciences.
Calmodulin
Phospholipase A2
Troponin
Calsequestrin
Calcineurin
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Phosphorylation of the insulin receptor on tyrosine side chains
Stimulation of cAMP synthesis through a G-protein
Inhibition of cAMP synthesis through a G-protein
Formation of IP3 and DAG
Activation of a guanylate cyclase
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The binding of calcium to transcription factors, which in turn bind to promoters and enhancers of genes
The activation of calmodulin-regulated protein kinases that phosphorylate nuclear transcription factors
The direct binding of calcium to the DNA double helix
The methylation of DNA in the promoters and enhancers of genes by a calcium-dependent enzyme
The binding of calcium to mRNA, which stabilizes the RNA against the action of RNA-degrading enzymes
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Phosphodiesterase
Protein kinase C
Protein kinase B
Phospholipase C
Ras
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Causes the pituitary gland to overproduce TSH
Prevents hydrolysis of bound GTP by the Gs protein
Prevents binding of TSH to the TSH receptor
Prevents the activation of phospholipase C by a G-protein
Prevents the activation of protein kinase A
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Inability of the Gs protein to hydrolyze its bound GTP
Inability of the Gi protein to hydrolyze its bound GTP
Inability of the protein kinase A regulatory subunits to respond to cAMP
Absence of the CREB (cyclic AMP response element binding) protein
Degeneration of the TSH-producing thyrotrophs in the anterior pituitary gland
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A voltage-gated ion channel
A ligand-gated ion channel
A cytoplasmic protein kinase
A hormone receptor that is linked to G-proteins
A hormone receptor with a tyrosine-specific protein kinase activity
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It phosphorylates proteins on tyrosine side chains
It is coupled to the Gi protein
It is coupled to the Gq protein
It has guanylate cyclase activity
It mediates many of its effects through the production of nitric oxide (NO)
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A calcium channel in the ER membrane
Phospholipase C
Protein kinase C
A nuclear DNA-binding protein
Calmodulin
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Neurotransmitters
Hormones acting through cGMP
Steroid hormones
Insulin
Hormones that are coupled to heterotrimeric G proteins
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Dephosphorylation of transcription factors by the protein phosphatase calmodulin
Binding of the Ras protein to the promoters and enhancers of genes
Phosphorylation of transcription factors by MAP kinases
Phosphorylation of a catabolite activator protein in the nucleus
Phosphorylation of the CREB protein on tyrosine side chains
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Nicotinic: has guanylate cyclase activity; muscarinic: ion channel
Nicotinic: ion channel; muscarinic: coupled to G-proteins
Nicotinic: coupled to G-proteins; muscarinic: has adenylate cyclase activity
Nicotinic: coupled to G-proteins; muscarinic: ion channel
Nicotinic: ion channel; muscarinic: has tyrosine-specific protein kinase activity
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Inhibits the endothelial nitric oxide synthase
Opens voltage-gated calcium channels in the plasma membrane of vascular smooth muscle cells
Blocks voltage-gated calcium channels in the plasma membrane of endothelial cells but not vascular smooth muscle cells
. Inhibits the Gs protein in vascular smooth muscle cells
Prevents the degradation of cAMP in vascular smooth muscle cells but not other cells in the body
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It is translocated to the nucleus where it binds to response elements in the regulatory sequences of the genes.
It triggers cAMP formation, and cAMP activates genes by binding to the catabolite activator protein
It releases calcium from the ER, which activates protein kinase C in the nucleus
It causes phosphorylation of nuclear transcription factors by protein kinase A
It induces the formation of the second messenger IP3, which activates transcription by binding to nuclear transcription factors
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