This EMT I Exam Practice focuses on medical emergencies, assessing critical skills like oxygenation, ventilation, and management of severe pulmonary diseases. It's designed for EMTs to refine their response strategies in emergency medical scenarios.
Perfusion
Oxygenation
Diffusion
Ventilation
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An accurate indicator of severe pulmonary disease
Not related to future ventilation needs
Pertinent to theis event; you should intubate this patient immediately
Important to the hospital staff but not important in the prehospital environment
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Asthma
Emphysema
Chronic bronchitis
Pneumonia
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Increasing mucus production
Preventing atelectasis
Increasing surfactant production
Dilating the bronchi
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Administer oxygen via a simple mask at 4 to 6 L per minute
Administer oxygen via a nasal cannula at 2 L per minute
Adminsiter high-flow oxygen and be prepared to ventilate if neccessary
Withhold oxygen therapy
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Hypertension
Excessively increased preload
Hypocapnia
Pulsus paradoxus
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Rales (crackles)
Rhonchi
Stridor
Wheezes
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Productive cough
Fever
Rhonchi
Rales
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Keep alveoli open
Ventilate patients with pneumothorax
Overcome upper airway obstruction
Open constricted bronchi
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Tension pneumothorax
Medical pneumothorax
Simple pneumothorax
Spontaneous pneumothorax
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Facial arteries
Subclavian atreries
External carotid arteries
Internal carotid arteries
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Mean arterial pressure and intracranial pressure
Diastolic blood pressure and intracranial pressure
Intracranial pressure and cerebral blood flow
Cerebral blood flow and systolic blood pressure
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Not be affected
Moderately increase
Decrease
Not be affected
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Aimed at maintaining a PCO2 of 30mm Hg
Withheld, allowing the carbon dioxide levels to return to normal
Increased until the patient stops posturing
Continued at a rate of more than 40 breaths per minute
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Decreased blood pressure, pulse, and respiratory rate
Elevated blood pressure, decreased pulse and respiratory rate
Decreased blood pressure, increased pulse and respiratory rate
Elevated blood pressure, pulse and respiratory rate
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Retrograde amnesia
Increased intracranial pressure
Hypoxia
Rapidly worsening level of consciousness
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Spastic rigidity
Flaccidity
Decerebrate posturing
Decorticate posturing
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Motor response
Cranial nerve evaluation
Level of consciousness
Pupillary response
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Metabolic
Alcoholic
Structural
Hypoglycemic
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I competes with narcotics at the receptor sites
It stimulates the brain stem
It depresses seizure activity
It stimulates teh sympathetic nervous system
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Injectable drugs
Barbituates and narcotics
Opiates
Sedatives and narcotics
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Hemorrhage and sepsis
Occlusion and hemorrhage
Occlusion and neoplasm
Neoplasm and sepsis
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Hemorrhagic stroke
Transient ischemic attack
Cerebral embolism
Cerebral thrombosis
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The only mechanism of a TIA is occlusive injury
Patients undergoing a TIA exhibit only weakness and paralysis
Patients undergoing a TIA have no real risk for having a stroke
The signs and symptoms of a TIA resolve within 24 hours
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Gripping an object in the hands causes the arms to drift
One arm drifts down compared to the other
The patient cannot hold his or her arms in front of the body for 3 minutes
Pressure applied against outstretched arms causes them to drift
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Rapidly transport the patient to an appropriate facility
Determine whether the stroke is hemorrhagic or occlusive
Administer glucose
Control seizure activity
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Caused by metabolic abnormalities
Secondary to infection
That occurs primarily in febrile children
With no known correctable or avoidable causes
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Clonic
Tonic
Tonic-clonic
Petit mal
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Petit mal
Clonic
Tonic-clonic
Tonic
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A period of abnormal perception prior to a seizure
Seizure activitiy on only one side of the body
Repetitive seizures without any period of awakening
The medical term for psychogenic epilepsy
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The patient is tachycardic
Tonic-clonic movements are observed
Unconsciousness last for several minutes
A feeling of lightheadedness before an episode
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Lymphatic system
Target tissues
Blood stream
Effector organs
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Pancreas
Liver
Stomach
Gallbladder
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Decrease glucose matabolism
Eliminate pancreatic glycogen stores
Transport glucose into the cells
Increase blood glucose concentration
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Normal
The high end of normal
Extremely elevated
Abnormally low
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Glucogen
Insulin
Epinephrine
Cortisol
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Inappropriate carbohydrate metabolism
Excessive prodution of glucagon
The liver's inability to break down glucose
Inadequate production of insulin
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Excessive carbohydrate intake
The liver's inability to store glycogen
Decreased production of insulin
Poor protein breakdown
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Create a metabolic alkalosis
Compensate for metabolic acidosis
Create a repiratory alkalosis
Reverse respiratory acidosis
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Thiamine
Dextrose
Insulin
Large boluses of normal saline
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Combative behavior
Warm, moist skin
Bradycardia
Flaccidity
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Cool, moist skin and kussmaul respirations
Wam, moist skin and hypertension
Warm, dry skin and kussmaul respirations
Cool dry skin and hypertension
Unresponsive patients
Teenage patients
Type I diabetics
Alcoholic patients
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Insulin
Glucagon
Thiamine
Flumazenil
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An IV line cannot be established
The patient is known to have liver disease
You suspect DKA
Blood glucose levels are below 100 mg/L
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Excretion of ketones
Breakdown of liver glycogen
Transport of glucose into the cells
Production of insulin
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Topical steriods
Antihistamines
Hot packs and elevation
Beta blockers
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Kinin
Heparin
Histamine
Leukotriene
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