Explore the role of neutrophils in inflammation through 'Path Inflammation Dr Johnson part 2'. This quiz assesses understanding of cellular responses in pathological conditions like myocardial infarction, focusing on neutrophil-driven processes such as chemotaxis and enzyme release.
Diapedesis
Exudation
Emigration
Anaphylaxis
Chemotaxis
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Venous congestion
Lymphatic obstruction
Increased arterial flow
Increased arterial pressure
Increased vascular permeability
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Activation of eosinophils
Degranulation of mast cells
Extrusion of enzymes from macrophages
Release of bacterial toxins
Release of hydrolases from neutrophils
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Plasma cells
Eosinophils
Vascular endothelial cells
Tissue mast cells
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Immature lymphocytes
Immatute band forms of PMNs
Increase in mature PMNs
A decrease in PLTs
An anemia
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Bradykinin
Complement
Endotoxin
Histamine
Tumor necrosis factor
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C1
C3a
C4
C5a
C5b67
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Neutrophil
Lymphocyte
Basophil
Eosinophil
Monocyte
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Tissue
Serum
Neutrophils
Lymphocytes
Plasma cells
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Nuclear division of granulocytes
Atypical regeneration of epithelium
Megakaryocytes
Fusion or nuclear division of macrophage
Multiplication of nuclei in surrounding fibrocytes
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Macrophages
Plasma cells
Basophils
Neutrophils
Lymphocytes
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Lymphokines
Histamine
Bradykinin
Complement
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Plasmin
Hageman factor (XIIa)
C1 esterase
Thrombin
Kininogen
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Arteries
Arterioles
Capillaries
Venules
Veins
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Histamine
Complement
Kinins
Serotonin
Hageman factor
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Basophil
Eosinophil
Lymphocyte
Macrophage
Neutrophils
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Atrophy
Inflammation
Regeneration
Repair
Resolution
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