Neuron Project Final Quiz
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This quiz will cover the concepts in Lesson 1, Lesson 2 and Lesson 3 of The Neuron Project website. Before attempting this quiz, go back and review any lesson or activity you wish. Feel free to use the Activity Aid to help you with the questions. Aim for 100%. Retry the quiz as many times as needed to achieve this result. When you are finished, click the NEXT button. Good luck!
- 1.
Gaps between the myelin sheath along an axon are called:
- A.
Nodes of Ranvier
- B.
Dendrites
- C.
Synapses
- D.
Nucleus
Correct Answer
A. Nodes of RanvierExplanation
Gaps between the myelin sheath along an axon are called Nodes of Ranvier. These nodes are crucial for the conduction of nerve impulses. They allow the electrical signals to jump from one node to another, which speeds up the transmission of information along the axon. The myelin sheath acts as an insulating layer, and the nodes of Ranvier are the exposed areas where the axon membrane is not covered. This allows for the efficient propagation of the electrical impulses along the axon.Rate this question:
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- 2.
The chemical that moves from the axon of one neuron across the synaptic gap to the dendrite of another neuron is called a:
- A.
Enzyme
- B.
Receptor
- C.
Neurotransmitter
- D.
Transporter protein
Correct Answer
C. NeurotransmitterExplanation
A neurotransmitter is a chemical that is released by a neuron and travels across the synaptic gap to bind to receptors on the dendrite of another neuron. This allows for the transmission of signals between neurons in the nervous system. Enzymes are not responsible for this process, as they typically facilitate chemical reactions rather than transmission of signals. Receptors are the proteins on the receiving neuron that bind to neurotransmitters. Transporter proteins are involved in the reuptake of neurotransmitters back into the neuron that released them, but they do not directly move the neurotransmitter across the synaptic gap.Rate this question:
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- 3.
Which of the following is a function of the myelin sheath?
- A.
Receives information from other neurons or from sense organs
- B.
Increases the speed at which nerve impulses travel along an axon
- C.
Releases neurotransmitters that can cross over to neighboring neurons
- D.
Controls all cellular activity
Correct Answer
B. Increases the speed at which nerve impulses travel along an axonExplanation
The myelin sheath is a protective covering that surrounds nerve fibers. Its main function is to insulate and increase the speed of nerve impulses as they travel along an axon. This is achieved by preventing the leakage of electrical signals and allowing the impulses to jump from one node of Ranvier to another, a process known as saltatory conduction. This efficient transmission of nerve impulses helps to ensure fast and accurate communication between neurons, enabling rapid responses in the body.Rate this question:
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- 4.
Part of the neuron that releases neurotransmitters into the synaptic cleft.
- A.
Axon hillock
- B.
Dendrite
- C.
Cell body
- D.
Axon terminal
Correct Answer
D. Axon terminalExplanation
The axon terminal is the part of the neuron that releases neurotransmitters into the synaptic cleft. Neurotransmitters are chemical messengers that transmit signals between neurons. The axon terminal is located at the end of the axon, which is the long fiber that extends from the cell body of the neuron. When an action potential reaches the axon terminal, it triggers the release of neurotransmitters into the synaptic cleft, where they can bind to receptors on the dendrites of the neighboring neurons, allowing for communication between neurons.Rate this question:
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- 5.
A disease that destroys the protective and insulating myelin covering the neurons in a process called demyelination.
- A.
Multiple Sclerosis
- B.
ALS
- C.
Progressive Bulbar Palsy
- D.
Primary Lateral Sclerosis
Correct Answer
A. Multiple SclerosisExplanation
Multiple Sclerosis is the correct answer because it is a disease that causes the destruction of the protective and insulating myelin covering the neurons, leading to demyelination. ALS, Progressive Bulbar Palsy, and Primary Lateral Sclerosis are not associated with the specific process of demyelination.Rate this question:
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- 6.
Neurotransmitters are stored in:
- A.
Receptors
- B.
Synaptic cleft
- C.
Synaptic vesicles
- D.
Post-synaptic neuron
Correct Answer
C. Synaptic vesiclesExplanation
Neurotransmitters are chemicals that transmit signals between neurons. They are stored in synaptic vesicles, which are small sacs located within the axon terminal of the presynaptic neuron. When an action potential reaches the axon terminal, these vesicles release the neurotransmitters into the synaptic cleft, the small gap between the presynaptic and postsynaptic neurons. From there, the neurotransmitters bind to receptors on the postsynaptic neuron, allowing for the transmission of the signal. Therefore, the correct answer is synaptic vesicles.Rate this question:
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- 7.
When neurotransmitters are released into the synaptic cleft they bind to:
- A.
The pre-synaptic neuron receptor sites
- B.
The post-synaptic neuron receptor sites
- C.
Re-uptake pumps
- D.
Axon terminal
Correct Answer
B. The post-synaptic neuron receptor sitesExplanation
When neurotransmitters are released into the synaptic cleft, they bind to the post-synaptic neuron receptor sites. This is because these receptor sites are specifically designed to receive and respond to neurotransmitters. By binding to these receptor sites, neurotransmitters can transmit signals from the pre-synaptic neuron to the post-synaptic neuron, allowing for communication between neurons. The binding of neurotransmitters to post-synaptic receptor sites can lead to various effects, such as the opening of ion channels or the activation of intracellular signaling pathways.Rate this question:
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- 8.
How does inactivation of neurotransmitters occur?
- A.
Neurotransmitters are degraded by being broken down by enzymes or reused by active re-uptake.
- B.
Neurotransmitters eventually dissolve on their own.
- C.
Neurotransmitters dissipate into the brain matter and become useless.
- D.
Neurotransmitters bind together so they are too big to fit into receptor sites and are rendered inactive.
Correct Answer
A. Neurotransmitters are degraded by being broken down by enzymes or reused by active re-uptake.Explanation
Neurotransmitters are degraded by being broken down by enzymes or reused by active re-uptake. This process allows for the efficient removal and recycling of neurotransmitters after they have transmitted their signals across the synapse. Enzymes break down the neurotransmitters into smaller molecules, while active re-uptake involves the reabsorption of neurotransmitters by the presynaptic neuron for future use. This inactivation process helps regulate the levels of neurotransmitters in the synaptic cleft and ensures proper neurotransmission.Rate this question:
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- 9.
Some medications for the treatment of Schizophrenia block Dopamine receptors. What does that indicate about the role of Dopamine in Schizophrenia?
- A.
There is too little Dopamine in the brains of Schizophrenic people.
- B.
The receptors are faulty for Dopamine in the brains of Schizophrenic people.
- C.
Schizophrenic patients cannot produce enough Dopamine.
- D.
There is too much Dopamine in the brains of Schizophrenic people. Drugs that block Dopamine receptors will decrease the action of Dopamine.
Correct Answer
D. There is too much Dopamine in the brains of Schizophrenic people. Drugs that block Dopamine receptors will decrease the action of Dopamine.Explanation
The fact that medications for the treatment of Schizophrenia block Dopamine receptors indicates that there is too much Dopamine in the brains of Schizophrenic people. By blocking the receptors, these medications decrease the action of Dopamine, which helps to alleviate the symptoms of Schizophrenia.Rate this question:
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- 10.
If a medication inhibits the re-uptake of a neurotransmitter, what does that mean?
- A.
It means that it will decrease the level of the neurotransmitter available in the synaptic cleft and therefore decrease its action.
- B.
It means that it will increase the level of the neurotransmitter available in the synaptic cleft and therefore increase its action.
- C.
It means that the re-uptake of the neurotransmitter will be more efficient.
- D.
It means that the re-uptake process will help facilitate more neurotransmitter storage.
Correct Answer
B. It means that it will increase the level of the neurotransmitter available in the synaptic cleft and therefore increase its action.Explanation
If a medication inhibits the re-uptake of a neurotransmitter, it means that it will increase the level of the neurotransmitter available in the synaptic cleft and therefore increase its action. Re-uptake is the process by which neurotransmitters are taken back up into the presynaptic neuron after they have been released into the synaptic cleft. By inhibiting this process, the medication allows more neurotransmitter to remain in the synaptic cleft, leading to increased stimulation of the postsynaptic neuron and enhancing the neurotransmitter's effects.Rate this question:
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- 11.
Depression, suicide, impulsive behavior, and aggressiveness all appear to involve certain imbalances in:
- A.
Serotonin
- B.
Acetylcholine
- C.
Dopamine
- D.
GABA
Correct Answer
A. SerotoninExplanation
Depression, suicide, impulsive behavior, and aggressiveness are all associated with imbalances in serotonin. Serotonin is a neurotransmitter that plays a crucial role in regulating mood, sleep, appetite, and behavior. Low levels of serotonin have been linked to depression and increased risk of suicide. Imbalances in serotonin can also lead to impulsive behavior and aggression. Therefore, it is likely that these mental health issues involve certain imbalances in serotonin.Rate this question:
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- 12.
The loss of this neurotransmitter in certain parts of the brain causes the muscle rigidity typical of Parkinson’s disease.
- A.
Serotonin
- B.
Norepinephrine
- C.
Dopamine
- D.
Glutamate
Correct Answer
C. DopamineExplanation
Dopamine is the correct answer because the loss of this neurotransmitter in certain parts of the brain is known to cause the muscle rigidity that is characteristic of Parkinson's disease. Dopamine is involved in the regulation of movement and its deficiency in the brain leads to the motor symptoms associated with Parkinson's disease, such as muscle stiffness and rigidity.Rate this question:
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- 13.
This substance delivers a sedative punch once it goes into the brain. It interacts with GABA receptors and drives them to be more inhibitory.
- A.
Ecstacy
- B.
Alcohol
- C.
LSD
- D.
Marijuana
Correct Answer
B. AlcoholExplanation
Alcohol is the correct answer because it is a substance that can deliver a sedative effect on the brain. It interacts with GABA receptors, which are responsible for inhibitory signals in the brain, and enhances their inhibitory activity. This leads to a decrease in brain activity and can result in sedation.Rate this question:
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- 14.
This substance binds to Serotonin receptors but not always in the same way. This substance can inhibit or excite these receptors. This is why it has complex sensory effects.
- A.
LSD
- B.
Cocaine
- C.
Heroin
- D.
Methamphetamine
Correct Answer
A. LSDExplanation
LSD is the correct answer because it is a substance that binds to serotonin receptors but not always in the same way. It can either inhibit or excite these receptors, leading to its complex sensory effects.Rate this question:
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- 15.
This substance binds to the opiate receptors. This mechanism turns off Dopamine inhibition and Dopamine gets released into the synapse. This generates feelings of sedation.
- A.
Alcohol
- B.
LSD
- C.
Cocaine
- D.
Heroin
Correct Answer
D. HeroinExplanation
Heroin is the correct answer because it is a substance that binds to the opiate receptors in the brain. This binding action turns off the inhibition of dopamine, a neurotransmitter responsible for feelings of pleasure and reward. As a result, dopamine is released into the synapse, leading to feelings of sedation and euphoria.Rate this question:
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Dec 14, 2023Quiz Edited by
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Nov 19, 2012Quiz Created by
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