7 MCQs (Only one correct answer)Each MCQ: 1 mark, Total mark: 7 (you will your result immediately by the web)Allowed time: 10 minGood Luckdr Mohammed Abel Gawad
The binding sites at the actin filaments are activated by Ca.
The sliding filament theory of contraction assumes that sliding of actin continues as long as Ca is attached to troponin C
The detachment of myosin heads from actin needs binding of ATP.
The transverse tubules (T.T.) contains Ca voltage gated channels
Hypertrophy develops as a result of forced repeated contractions.
The function of the T system is the rapid transmission of the action potential from the cell -membrane to inside the muscle fibre
After the spike potential, the muscle excitability is abolished
Acetylcholine released at the M.E.P. is rapidly hydrolyzed by cholinesterase.
Inward spread of the depolarization wave along the transverse tubules.
Pumping of Ca back into the sarcoplasmic reticulum.
Release of Ca from the sarcoplasmic reticulum.
Binding of Ca to troponin C.
In isometric contraction, the elastic elements are not stretched.
During isometric contraction a load is moved and work is done.
During isotonic contraction most of the energy transformed into heat.
During isotonic contraction the muscle length is not changed.
The initiation of contraction occurs by binding of Ca to tropomyosin.
The action potential follows the work done.
The one way conduction at the M.E.P. is due to location of the chemical mediator (acetylcholine) in the nerve terminals.
Fatigue sets in after fatigue occurs in the nerves.
There is increased muscle excitability
Skeletal muscle responds to both faradic and galvanic current
Skeletal muscle has longer chronaxie
ACC is lower than CCC
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