Comprehensive exam covering cardiac anatomy, physiology, conditions, assessment, and management, suitable for medical education.
Both the fibrous and parietal serous pericardium are supplied by phrenic
It is attached to the sternum, respiratory diaphragm and great vessels of the heart
Its parietal and visceral serous layers form the pericardial cavity
Both the parietal and visceral serous pericardium are supplied by the vagus nerve
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Is the pacemaker of the heart
Is known as the parietal pericardium
Is the visceral pericardium
Lines the heart chambers
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Epicardium, myocardium
Epicardium, endocardium
Myocardium, endocardium

Endocardium, connective tissue
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Area on the 6th ICS
L atrium
Mitral pulse
L ventricle
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Coronary sinus

Superior vena cava
Inferior vena cava
Pulmonary veins
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R aortic sinus at the base of ascending aorta
L aortic sinus at the base of ascending aorta
R aortic sinus at the base of the arch of aorta
L aortic sinus at the base of the arch of aorta
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Musculi pectinati
Chordae tendinae
Trabeculae carneae
The skeleton of the heart
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Gap junctions
Transverse tubules
Sarcoplasmic reticulum
Intercalated discs
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SA node
Bundle of His and its branches
Purkinje fibers
AV node
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CN XII
CN XI
CN X
NOTA
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Foramen ovale
Fossa ovalis
Annulus ovalis
Ductus arteriosus
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5th left ICS near sternal border
Right lower end of the sternum
2nd L ICS near sternal border
2nd R ICS near medial end of the costal cartilage
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1,2,3,4
2,3,1,4
3,1,2,4
3,4,1,2
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The heart will stop. 

The ventricles will contract, but the atria will stop.
Another part of the heart, possibly the AV node, will become the 
pacemaker.
The heart will beat faster. 

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Cardiac muscle has more sarcoplasmic reticulum than skeletal muscle. 

Cardiac muscle has a prolonged period of slow repolarization called the plateau phase. 

Cardiac muscle has a shorter refractory period than skeletal muscle. 

Depolarization of cardiac muscle occurs when K+ and Na+ diffuse 
into the cell.
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Voltage-gated Ca2+ ion channels open
Voltage-gated K+ ion channels open
Voltage-gated Na+ ion channels close
Both B and C
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Ca2+ ions
K+ ions
Na+ ions
Cl- ions
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Depolarization of the atria
Repolarization of the atria
Depolarization of the ventricles
Repolarization of the ventricles
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Ectopic action potentials in the atria
Damage to the tricuspid valve
Excessive sympathetic stimulation
Excessive parasympathetic stimulation
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Closed, closed
Closed, open
Open, closed
Open, open
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65 mmHg
70 mm Hg
75 mmHg
80 mm Hg
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120 mm Hg
100 mm Hg
80 mm Hg
60 mm Hg
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Stroke volume
Preload
Cardiac output
Force of contraction
AOTA
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Option 1
Option 2
Option 3
Option 4
Plaques obstruct the vein
Plaques obstruct the artery
Blood clots form outside the vessel wall
Hardened vessels dilate to allow the blood to flow through
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Cardiogenic shock
Heart failure
Arrhythmias
Pericarditis
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Prinzmetal’s angina
Cardiac Syndrome X
Kounis Angina
Microvascular Angina
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Chest pain
Dyspnea
Edema
Palpitations
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Beta-adrenergic blockers
Calcium channel blockers
Narcotics
Nitrates
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Aortic
Mitral
Pulmonic
Tricuspid
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Abdominal aorta
Ascending aorta
Descending thoracic aorta
Arch of the aorta
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Lower back pain, increased BP, decreased RBC, increased WBC
Severe lower back pain, decreased BP, decreased RBC, increased WBC
Severe lower back pain, decreased BP, decreased RBC, decreased WBC
Intermittent lower back pain, decreased BP, decreased RBC, increased WBC
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Group A streptococcal infection
Group B staphylococcal infection
Actinobacteria
Entameba Histolytica
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Rheumatic Heart Disease
Patent Ductus Arteriosus
Tetralogy of Fallot
Congestive Heart Failure
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Exertional Dyspnea
Orthopnea
Cyanosis
Distended jugular veins
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Transposition of Great Vessels
Coarctation of the aorta
Total Anomalous PVR
Both A and C
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Transposition of Great Vessels
Tetralogy of Fallot
Total Anomalous PVR
Truncus Arteriosus
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Prolonged P-R interval
Inverted T wave
Absent QRS complex
All of the above
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Increase body temperature
Adrenergic stimulation of the heart
Sympathetic stimulation of the heart
Cholinergic stimulation of the heart
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Ach
N.Epinephrine
Dopamine
Glycine
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P wave
QRS complex
Q wave
T wave
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1st degree AV Heart Block
2nd degree AV Heart Block
3rd degree AV Heart Block
4th degree AV Heart Block
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Resistance training 1 day after PTCA
UE ergometer 5 days after sternotomy
Resistance training 1 month after CABG
Mild stretching of pectorals 2 days after CABG
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Ejection fraction is typical of a heart failure.
Ejection fraction is normal
Cardiac output is higher than normal
Cardiac ouput is normal
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Defer. Low Hgb.
Defer. Low Hct.
Defer. Low WBC.
Defer. Low platelets.
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I
II
III
IV
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Increasing both preload and SV
Decreasing both preload and SV
Increasing preload, decreasing SV
Decreasing preload, increasing SV
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ST elevation
T –wave inversion
Decreased J-point
ST depression
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