The ECG shows a regular rhythm at a rate of 110 bpm. There is a P wave before each QRS complex, with a stable PR interval
(0.16 sec). The P wave is positive in leads I, II, aVF, and V4 -V6, and it has a normal duration (0.10 sec). Hence this is a sinus tachycardia ( not SVT).
The P waves are abnormal, and they are tall, narrow, and peaked in leads II, aVF, and V1-V2. The P-wave morphology is characteristic of a P pulmonale, a result of right atrial hypertrophy. This may also be termed a right atrial abnormality (RAA.)
The axis is rightward, between +90° and +180° (negative QRS complex in lead I and positive QRS complex in lead aVF).
The major finding is a tall R wave in lead V1 , defined as an R wave taller than 7 mm (seven small boxes) or an R /S > 1. The
tall R wave in lead V1 along with the rightward axis and a P pulmonale (right atrial hyper trophy) are characteristic of right ventricular hypertrophy (RVH).
The criteria for the diagnosis of RVH include:
• R-wave amplitude (in mm) in lead V1 > 7 mm
• R/S ratio in lead V1 > 1
• R/S ratio in lead V6 (or V5)
Supporting criteria for RVH include:
• Right axis deviation (between +90° and +180°), which is diagnosed by a QRS complex that is negative in lead I and positive in lead aVF
• Right atrial hypertrophy (P pulmonale); the P wave is tall (> 0.25 mV), narrow (
• Associated ST-segment depression and T-wave abnormalities in leads V1-V3
The combination of RVH and right atrial hyper t rophy on the ECG and a loud P2 on exam suggests the presence of elevated pulmonary pressures. Pulmonary arterial hypertension is clinically associated with scleroderma and is the most likely diagnosis.