This quiz focuses on medications used to treat hypertension, exploring common side effects, specific drug properties, and patient scenarios related to these medications.
ACE inhibitors
Beta-blockers
Antibiotics
Diuretics
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Hypokalemeia
Hyperkalemia
Hyponatremeia
Hypernatremia
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ACE inhibitor
Calcium Channel Blocker
K sparing diruretic
Loop Diuretic
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Hypokalemia
Hypercalcemia
Hyperkalemia
Hyperphosphemia
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Lisa will need to go on a K supplement.
Lisa has become hyperkalemic and needs reeducation on her medication.
Lisa is only showing 3 of the 5 indicators for hyperkalemia, so she needs to continue her medication.
Lisa needs to be put on a different hypertensive because she is complaining too much.
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It releases Ca and HCO3 while holding onto K.
It blocks the angiotensin I from turning into angiotensin II, leaving the vessels dilated for better blood flow.
It blocks the reabsorbtion of Na.
Blocks stimulation of beta .
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Lasix
Aldactone
Prinivil
Lostran
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Losartan
Zestril
Atenolol
Norvasc
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Lasix
Aldactone
Lisinopril
Prazosin
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Decrease in K
Increase bradykin
Has the same side effects as ACE inhibitors
Renal Failure
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There are no common side effects.
It is possible to have angioedema
It is possible for renal failure.
It's less likely to increase K
All the above
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It may cause impotence.
I could have CHF.
I hate headaches.
I don't want dry mouth.
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Checks pupil dilation
Checks apical pulse
Check labs for K wasting
Check for peripheral edema
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It keeps norephinephrine from stimulating the muscle walls of the small arteries and veins.
It blocks beta 1 and causes vasodilation and decreases HR.
It helps maintain renal function.
Increases excretion of H2O and Na.
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Loop diuretic
Beta Blocker
ACE inhibitor
Calcium Channel Blocker
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I may have headaches
It causes vadodilation and decreases SVR and lowers B/P
It may cause edema peripherally
All the above
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HR
Dry Coughing
Pt becomes hypotensive
If K levels increase
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Dry mouth
Increased K
Orthostatic BP w/first dose
Palpitations
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Ephinephrine
Angiotensin 1
Norepinephrine
PTH
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Over abundance of K in her system
Orthostatic BP problems
Drug Induced lupus syndrome
Withdrawl phenomenon
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Increasing parasympathetic response
Keeps the hormone norepinephrine from stimulating the muscle walls of the small arteries/veins.
Decreases sympathetic response
Directly inhibits renin.
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Decreases sympathetic response
Peripheral vasodilation
Directly inhibits renin
Blocks Ca from stimulating muscle walls
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Increases HR
Na retention
Drug induced lupus syndrome
All the above
None of the above
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Apresoline
Clonidine
Tekturna
Praosin
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Quiz Review Timeline (Updated): Jan 15, 2025 +
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