Explore the role of calcium channel blockers in medical treatment through this quiz. Assess knowledge on their classification, applications in disease management like hypertension and angina, and understanding of their mechanism. Essential for students and professionals in medicine and pharmacology.
True
False
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Excessive vasodilation
Excessive vasoconstriction
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Contration, heart rate, and conduction
Neurotransmitter release
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True
False
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True
False
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True
False
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Relaxation
Contraction
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True
False
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True
False
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Contraction
Relaxation
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Decreased heart rate, decreased conduction
Increased heart rate, increased conduction
Verapamil and diltiazem are not able to decrease the rate of recovery
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Nitrates
Penylalkylamines
Dihydropyridines
Benzothiazepines
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Nicardipine
Amlodipine
Diltiazem
Nifedipine
Verapamil
Felodipine
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True
False
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L
S
N
T
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Diltiazem
Nifedipine
Verapamil
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N
T
L
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Dihydropyridines (Nifedipine)
Phenylalkylamines (Verapamil)
Benzothiazepines (Diltiazem)
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True
False
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True
False
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Vasospastic angina
Stable exertional angina
Essential hypertension
All of the above
None of the above
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True
False
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True
False
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Essential hypertension
Cardiac arrhythmias
Type II Diabetes
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Decrease pressure and decreased cardiac preload
Increased pressure and increase cardiac preload
Decreased pressure and decreased cardiac afterload
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More calcium enters the cell
Negative inotropic effect
Less calcium enters the cell
Positive inotropic effect
Decrease in contractility
Increase in contractility
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Nifedipine
Diltiazem
Verapamil
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Nifedipine
Verapamil
Diltiazem
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Inhibits myosin and actin's interactions to produce contraction
Allows myosin and actin's interactions to produce contraction to occur uninhibited
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Phase 0
Phase 1
Phase 2
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True
False
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Verapamil IV
Diltiazem IV
Nifedipine IV
A and B
B and C
A and C
A, B, and C
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Their effectiveness is dependent on the frequency of use of the drugs
Their effectiveness is dependent on the frequency of use of the channels
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Resting
Inactivated
Activated
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Cardiac muscle cells use calcium to bind troponin C
Vascular smooth muscle cells use calcium to bind troponin C
Cardiac muscle cells use calcium to bind calmodulin
Vascular smooth muscle cells use calcium to bind calmodulin
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More potent vasodilatory than verapamil or dihydropyridines
Less potent vasodilatory than verapamil or dihydropyridines
In vasodilatory doses, less direct negative chronotropic and dromotropic effects than verapamil
In vasodilatory doses, more direct negative chronotropic and dromotropic effects than verapamil
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Calcium binds calmodulin
Calcium binds troponin C
The interaction of actin with myosin to produce contraction is inhibited
The interaction of actin with myosin to produce contraction occurs uninhibited
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Calmodulin activates myosin LC kinase to phosphorylate myosin LC
Calcium binds to calmodulin
Calmodulin inhibits myosin LC kinase, so myosin LC cannot be phosphorylated
Phopshorylated myosin LC interacts with actin
Contraction occurs because phosphorylated myosin LC interacts with actin
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Widespread in cardiovascular system
Activated when the cell is depolarized
Large conductance of long lasting duration
Inhibited by calcium channel blockers
Responsible for the upstroke of the cardiac action potential
Responsible for the plateau phase of the cardiac action potential
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Dihydropyridines have a preferentially selective effect on blood vessel calcium channels
Dihydropyridines do not effect the rate of recovery of the calcium channels
Dihydropyridines can show effects on heart rate and conduction
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Diltiazem
Verapamil
Both
Neither
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Long lasting current
High conductance
Large channels
Enriched in the neurons
Enriched in cardiac and smooth muscle, in the SA node and Purkinje cells
Blocked by spider and snail toxins
Blocked by calcium channel blockers
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Large conductance
Small conductance
Intermediate conductance
Blocked by calcium channel blockers
Blocked by spider and snail toxins
Enriched in neurons
Enriched in cardiac muscle
Play a role in neurotransmitter release
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Longer period of inactivation
Prevention of cycling of the channel
Decreased refractory period
Increased refractory period
Slows heart rate
Increases conduction
Decreases conduction
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