How Much Do You Know About Antidyslipidemic?
Dyslipidemia is the increase in amounts of lipids or fat within the body. This situation increases the chance of clogged arteries and therefore, many circulatory concerns such as stroke. There are some drugs given out for patients to curb this. How much do you know about Antidyslipidemic? Well take up the short quiz below and get to find out for sure.
- 1.In patients with combine hyperlipidemia. Which of the following drugs is most likely to increase this patient’s triglyceride and VLDL cholesterol concentrations when used as monotherapy?
- A.
Atorvastatin
- B.
Cholestyramine
- C.
Ezetimibe
- D.
Gemfibrozil
- 2.Which of the following is a major mechanism of gemfibrozil’s action?
- A.
Increased excretion of bile acid salts
- B.
Increased expression of high-affinity LDL receptors
- C.
Increased secretion of VLDL by the liver
- D.
Increased triglyceride hydrolysis by lipoprotein lipase
- 3.Which of the following is a major toxicity associated with gemfibrozil therapy?
- A.
Bloating and constipation
- B.
Cholelithiasis "gall stones"
- C.
Hyperuricemia
- D.
Liver damage
- 4.In a patient with dyslipidemia and has a history of gout, which of the following drugs is most likely to exacerbate this condition?
- A.
Colestipol
- B.
Ezetimibe
- C.
Gemfibrozil
- D.
Niacin
- 5.While treating dyslipidemia, after being counseled about lifestyle and dietary changes, the patient was started on atorvastatin. During his treatment with atorvastatin, it is important to routinely monitor serum concentrations of which of the following?
- A.
Blood urea
- B.
Alanine and aspartate aminotransferase
- C.
Platelets
- D.
Red blood cells
- 6.Six months after beginning atorvastatin, a patient’s total and LDL cholesterol concentrations remained above normal, his physician decided to add ezetimibe. Which of the following is the most accurate description of ezetimibe’s mechanism of an action?
- A.
Decreased lipid synthesis in adipose tissue
- B.
Decreased secretion of VLDL by the liver
- C.
Decreased gastrointestinal absorption of cholesterol
- D.
Increased endocytosis of HDL by the liver
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