Acute respiratory failure when it occurs can be either hypoxemic or hypersonic. As we just covered acute respiratory failure in class today there will be a text in some time. The quiz below is designed to check what we have covered and help you note areas to polish up on. Give it a try and all the best!
Sudden life-threatening deterioration of gas exchange in the lungs
Non-cardiac pulmonary edema with increasing hypoxemia despite treatment with O2
Sudden life-threatening pulmonary edema that causes a deterioration of gas exchange despite treatment with O2
Ventilatory failure
Atelectasis
Oxygenation failure
None of the above
Perfusion is normal, but ventilation is inadequate
Air morvement in/out of the lungs is the problem
Less oxygen in the alveoli
Low CO2 levels in blood
None of the above
All of the above
Pulmonary embolism
Pneumo/hemothorax
Kyphoscoliosis
Damage to the respiratory control center in the brain
Decreased function of respiratory muscles
None of the above
Movement of air is adequate
It can be caused by a PE
Perfusion is inadequate
None of the above
Confusion
Lethargy
Dyspnea
Anxiety
Diaphoresis
Accessory muscle use
Restlessness
Tachycardia
Decreased breath sounds
Elevated BP
Cyanosis
Respiratory arrest/failure
Confusion
Lethargy
Dyspnea
Anxiety
Diaphoresis
Accessory muscle use
Restlessness
Tachycardia
Decreased breath sounds
Elevated BP
Cyanosis
Respiratory arrest/failure
Sudden life-threatening deterioration of gas exchange in the lungs
Non-cardiac pulmonary edema with increasing hypoxemia despite treatment with O2
Sudden life-threatening pulmonary edema that causes a deterioration of gas exchange despite treatment with O2
Dyspnea
Myasthenia Gravis
Refractory hypoxemia
Cyanosis
Dense pulmonary infiltrates on CXR
Decreased pulmonary compliance
Non-cardiac pulmonary edema
Chest pain
Fluids shift into the alveoli, the alveoli and bronchii collapse, and lose lung compliance
The lung starts to repair itself; this is where the patient starts to get better or the condition deteriorates.
Fibrous tissue forms and lungs don't expand well; the effort to breathe increases O2 demand which causes more effort to breathe.
Fluids shift into the alveoli, the alveoli and bronchii collapse, and lose lung compliance
The lung starts to repair itself; this is where the patient starts to get better or the condition deteriorates.
Fibrous tissue forms and lungs don't expand well; the effort to breathe increases O2 demand which causes more effort to breathe.
Fluids shift into the alveoli, the alveoli and bronchii collapse, and lose lung compliance
The lung starts to repair itself; this is where the patient starts to get better or the condition deteriorates.
Fibrous tissue forms and lungs don't expand well; the effort to breathe increases O2 demand which causes more effort to breathe.
Retractions - not always (tissues between ribs and above sternum pull in)
Dyspneic
Non-productive cough
Accessory muscle used
Pallor or cyanosis
Significant CXR changes; pulmonary infiltrates
Restlessness
CXR clear
Respiratory alkalosis
Respiratory acidosis
Retractions - though not always (tissues between ribs and above sternum pull in)
Dyspneic
Non-productive cough
Accessory muscle used
Pallor or cyanosis
Significant CXR changes; pulmonary infiltrates
Restlessness
CXR clear
Respiratory alkalosis
Respiratory acidosis
O2 therapy
Maintain IV volume and IV access
Ambulation
Treat cause
Conserve energy
ABGs
Monitor vitals
Glucocorticoids
Antibiotics
Pulse Ox
Turn regularly
Treatment-resistant hypoxemia
Refractory hypoxemia
Refractory Hemo-deoxygenation
True
False