There are different ways that one can review the materials they have learnt in class and this can either be through study groups, checking past tests or looking for practice questions. The study guide quiz below is the third in a series of tests designed to adequately prepare you for the pharmacology exam. Why don’t you give it a shot and see just how high you will score?
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False
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Diabetic ketoacidosis
Nonketotic coma
Diabetic retinopathy
Hypertension
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Osteoblasts; osteoclasts
Vitamin D; vitamin E
Parathyroid hormones; GnRH
Epinephrine; norepinephrine
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False
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70-130 mg/dL
10-60 mg/dL
150-220 mg/dL
240-300 mg/dL
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False
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Glucocorticoids
Mineralocorticoids
Androgens
Inuslin
1α-hydroxylase
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Primary hypothyroidism
Secondary hypothyroidism
Tertiary hypothyroidism
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False
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Pancreatic β cells
Pancreatic α cells
Follicular cells
Pancreatic δ cells
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False
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Actos
Avandia
Glipizide
Metformin
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False
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False
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Insulin receptor binds insulin and undergoes autophosphorylation on its carboxyl-terminal Tyr residues.
Insulin receptor phosphorylates IRS-1 on its Tyr residues.
SH2 domain of Grb2 binds to P-Tyr of IRS-1. Sos binds to Grb2, then to Ras, causing GDP release and GTP binding to Ras.
Activated Ras binds and activates Raf-1.
Raf-1 phosphorylates MEK on two Ser residues, activating it. MEK phosphorylates ERK on a Thr and a Tyr residue activating it.
ERK moves into the nucleus and phophorylates nuclear transcription factors such as Elk1, activating them.
Phosphorylated Elk1 joins SRF to stimulate the transcription and translation of a set of genes needed for cell division.
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PI-1 Kinase
PI-2 Kinase
PI-3 Kinase
PI-4 Kinase
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Hyperglycemia
Hypoglycemia
Lipodystrophy
Weight gain
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Estradiol (E2)
Progesterone (P4)
GnRH
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False
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Albumin
Thyroxine-binding globulin (TBG)
GLUT2
Hemoglobin
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False
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False
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Bind reversibly to 50S ribosomal subunits
Inhibit the translocation step
Cause conformational change that terminates protein synthesis by interfering with transpeptidation
All of the above
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False
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Bacteriostatic; Bactericidal
Bactericidal; Bacteriostatic
Macrophage; Dendrites
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False
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Levothyroxine sodium
Liothyronine sodium
Methimazole
Carbimazole
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Insulin receptor phosphorylates IRS-1 on its Tyr residues.
Phosphorylated Elk1 joins SRF to stimulate the transcription and translation of a set of genes needed for cell division.
ERK moves into the nucleus and phophorylates nuclear transcription factors such as Elk1, activating them.
Raf-1 phosphorylates MEK on two Ser residues, activating it. MEK phosphorylates ERK on a Thr and a Tyr residue activating it.
Insulin receptor binds insulin and undergoes autophosphorylation on its carboxyl-terminal Tyr residues.
SH2 domain of Grb2 binds to P-Tyr of IRS-1. Sos binds to Grb2, then to Ras, causing GDP release and GTP binding to Ras.
Activated Ras binds and activates Raf-1.
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Pancreatic F cells
Pancreatic β cells
Pancreatic δ cells
Pancreatic α cells
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False
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False
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Corticosteroids
Androgens
Glucagon
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Immune function
Renal function
Liver function
Blood flow/perfusion
Age
Pregnancy
Lactation
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Chloramphenicol
Tetracycline
Macrolides
Clindamycin
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Quiz Review Timeline (Updated): Mar 22, 2023 +
Our quizzes are rigorously reviewed, monitored and continuously updated by our expert board to maintain accuracy, relevance, and timeliness.
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