Based off chaper 18-19 of Nancy Caroline's emergency medical care in the streets
Hypotension
Low cardiac output
An increase in preload
Decreased coronary perfusion
Localized myocardial ischemia
Any condition that causes increased preload
Decompensated congestive heart failure
Damage to more than 10% of the left ventricle
Dehydration
External bleeding
Diabetic ketoacidosis
Plasma loss from burns
Postural syncope when standing up
A furrowed tongue or shrunken eyes
Tenting of the skin when it is pinched
A decreased pulse rate upon standing
Profound vomiting and diarrhea cause by bacterium
Increased microvascular permeability and capillary leakage
Widespread vasoconstriction and preferential blood shunting
Persistent fever that results in internal loss of body fluids
An absence of sweating
A rapid, bounding pulse rate
Cool, clammy, and pale skin
Hemiplegia or hemiparalysis
Experiences urticaria and rhinorrhea after exposure to an allergen
Reacts violently to a substance to which he or she has been sensitized
Experiences a mild allergic reaction after initial exposure to an allergen
Develops a heightened reaction to a substance deemed foreign by the body
The precapillary sphincters remain constricted
Oxygen cannot diffuse across the cellular membrane
The postcapillary sphincters remain constricted
Of aerobic metabolism and carbon dioxide production
Inhibits hemoglobin in the red blood cells from binding with and carrying oxygen
Results in potent vasoconstriction and constriction of the postcapillary sphincters
Immediately depresses respiratory system function, resulting in severe hypoxemia
Causes a decrease in hydrogen ion production and an increase in the pH of the blood
Noncardiogenic pulmonary edema
Increased lactate metabolism by the liver
Marked decrease in cardiac ejection fraction
Decreased surfactant production by the alveoli
The patient's level of consciousness is a poor indicator of perfusion, because he or she is generally restless
An increase in respiratory rate and depth creates a compensatory respiratory alkalosis to offset metabolic acidosis
Chemical mediators released by the autonomic nervous system result in significant increase in blood pressure
Pulse pressure widens as the sympathetic nervous system releases catecholamines in response to poor perfusion
Anxiety or agitation
Dilation of the pupils
Absent peripheral pulses
Response to painful stimuli
A marked increase in cardiac afterload
A systolic BP that is less than 90 mm Hg
Vascular dilation and decreased blood flow
Sympathetic nervous system compensation
Establishing a patient airway
Forming a general impression
Vascular dilation and decreased blood flow
Sympathetic nervous system compensation
Pale, cool, clammy skin
Sluggishly reactive pupils
An altered mental status
An increased diastolic blood pressure
Applying warm blankets
Controlling major bleeding
Establishing a patent airyway
Following BSI precautions
In a semi-sitting position
Supine with the legs elevated
In the trendelenburg position
On the left side with the legs elevated
They remain in the vascular compartment for up to 4 hours
You should give 2-3 times the volume of blood lost
They improve tissue perfusion by carrying oxygen to the cells
20% of the solution quickly diffuses out of the intravascular space
Administer a 1000 mL crystalloid bolus if the patient's lungs are clear
Dopaine infusions greater than 10 mcg/kg/min often cause bradycardia
Norepinephrine is the vasopressor of choice for prehospital blood pressure support
Prolonged efforts to stabilize the patient in the field are not recommended
Diphendydramine
Epinephrine
Ipratropium
Albuterol
Antidepressants
Acetaminophen
Antihypertensives
Corticosteroids
An obese patient
A bedridden patient
A hypertensive patient
A patient with diabetes
8
10
12
18
Tetanus
Gangrene
Fasciitis
Lymphedema
Inflammation of the injured blood vessels
Ruptured of large blood vessels in the dermis
Aggregation of platelets to the injured site
Leakage of fluid into spaces between the cells
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