What do you know about liver cirrhosis? Try these liver cirrhosis MCQs with answers, and find out how well you know about the disease. Basically, cirrhosis is a very late stage of fibrosis of the liver caused by chronic alcoholism. The damage caused to the liver by cirrhosis can't be undone, but they can stop further damage if the patient wants. With this quiz, you can learn several new things as well. All the best for a perfect score! Also, share this liver cirrhosis quiz with your friends to see what is their understanding of liver cirrhosis.
Dyspnea and fatigue
Ascites and orthopnea
Purpura and petechiae
Gynecomastia and testicular atrophy
Diaphoresis and tremors.
Increased blood pressure and heart rate.
Illusions.
Delusions of grandeur.
Pork
Milk
Chicken
Broccoli
Alcoholic cirrhosis
Postnecrotic cirrhosis
Biliary cirrhosis
Compensated cirrhosis
Pressure in the portal vein
Production of serum albumin
Secretion of bile salts
Interstitial osmotic pressure
lactulose (Chronulac)
ethacrynic acid (Edecrin)
folic acid (Folvite)
thiamine (Vitamin B1)
Chronic low serum protein levels result in inadequate tissue repair, allowing the esophageal wall to weaken.
The enlarged liver presses on the diaphragm, which in turn presses on the esophageal wall, causing the collapse of blood vessels into the esophageal lumen.
Increased portal pressure causes some of the blood that normally circulates through the liver to be shunted to the esophageal vessels, increasing their pressure and causing varicosities.
The enlarged liver displaces the esophagus toward the left, tearing the muscle layer of the esophageal blood vessels, which allows small aneurysms to form along the lower esophageal vessels.
“The low protein diet will result in reduced clotting.”
“The increased production of bile decreases clotting factors.”
“The liver affected by cirrhosis is unable to produce clotting factors.”
“The required medications reduce clotting factors.”
Deflate the esophageal balloon.
Monitor VS
Encourage him to take deep breaths.
Notify the MD
Esophageal varices
Ascites
Peripheral edema
Asterixis (liver flap)
Decreased red blood cell count
Decreased serum acid phosphate level
Elevated white blood cell count
Elevated serum aminotransferase
Impaired clotting mechanism
Varix formation
Inadequate nutrition
The trauma of the invasive procedure
Increased urine output
Altered level of consciousness
Decreased tendon reflex
Hypotension
Expanding ecchymosis
Ascites and serum albumin of 3.2 g/dl
Slurred speech
Hematocrit of 37% and hemoglobin of 12g/dl
Dry skin
Hepatomegaly
Peripheral edema
Pruritus
Conversion of excess glucose into glycogen for storage
Conversion of conjugated bile to unconjugated bile
Regulation blood coagulation
Production of bile salts
Conversion of ammonia to urea
8-13cm
2-3cm
1 foot
6-12cm
Medial and lateral lobes
Anterior and posterior halves
Superior and inferior halves
Left and right parts of the left lobe
Right and left hypochondrium
Epigastrium and some left hypochondrium
Right hypochondrium and epigastrium
Epigastrium and umbilicus
True
False
Posterior and superior to the right lobe
Anterior and lateral to the left lobe
Posterior and lateral to the left lobe
Inferior to the left lobe
True
False
Falciform ligament
Right hepatic vein
Ligament teres
Portal vein
2
4
6
8
Falciform ligament
Ligament teres
Ligament venosum
Subphrenic ligaments
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