Explore the mechanisms of muscle function in 'Block 3 Cardiac and Smooth Muscle Function'. This quiz covers the dynamics of cardiac and smooth muscle contraction and relaxation, including molecular interactions and physiological responses crucial for medical and biomedical students.
To increase the extracellular Ca2+ concentration
To increase Ca2+ entry through DHP receptors
To increase Ca2+ release through ryanodin receptors
To decrease the extracellular Ca2+ concentration to zero
To block the activity of Ca2+/3Na+ exchanger
Troponin C
Calsequestrin
Tropomycin
Calmodulin
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Thick and thin filaments arranged in sarcomeres
Troponin
Elevation of intracellular[Ca++] for excitation-contraction coupling
Spontaneous depolarization of the membrane potential
High degree of electrical coupling between cells
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Simple diffusion
Facilitated diffusion
Secondary active transport
Endocytosis
Primary active transport
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Action potential and contraction are of similar duration
Autonomic nerve stimulation is not frequent enough
Lnsufficlent Cl is released from the sarcoplasmic reticulum
Return of Ca 2' to the sarcoplasmic reticulum by Ca-ATPase is too fast
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Lidocaine, a Class I antiarrhythmetic, voltage-gated Na+ channel blocker
Sympathetic stimulation
Ibutilide, a Class III antiarrhythmetic, that activates the (If) slow Na' channels
Diltiazem, a class IV antiarrhythmetic, voltage-gated Ca+ channel blocker
A large cup of caffeinated coffee
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Dephosphorylation of myosin molecules
Phosphorylation of troponin molecules
Movement of tropomyosin over myosin binding sites
Storage of all of the calcium ions within the sarcoplasmic reticulum
Restoration of normal resting membrane potential
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Cardiac ventricular muscle
Cardiac AV nodal tissue
Skeletal muscle fibers
Nerve cell bodies
Presynaptic nerve terminals
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G-protein linked
Ligand gated mixed cationic channels
Ligand gated cl channels
Voltage gated mixed cationic channels
ATPdependent Ca 2-pumps
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Actin is phosphorylated
Calcium binds to tropomyosin.
Extracellular calcium is required
Regulation is on the thick filament
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An increase in afterload causes an increase in shortening velocity
For a given afterload, an increase in preload leads to an increase in shortening velocity
A decrease in afterload causes a decrease in shortening velocity
For a given shortening velocity, an increase in preload causes a decrease in afterload
For a given preload, an increase in afterload causes an increase in shortening velocity
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