CTL Week 8 - Radiology of the GI system
CTL Week 8 - Miscellaneous GI pathology
CTL Week 8 - Hepatitis
Diaphragmatic hernia
Pyloric stenosis
Esophageal atresia
Esophageal rings
Gastric heterotopia in the duodenum
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Irritable bowel syndrome
Clostridium difficile
Celiac disease
Colorectal carcinoma
Clostridium botulinum
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Entamoeba hystolitica
Vibrio cholera
Shigella
Salmonella typhi
Entero-hemorrhagic E coli (EHEC) O157:H7
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Congenital duodenal atresia
Pyloric stenosis
Tracheo-esophageal fistula
Mallory-Weiss syndrome
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Hemachromatosis
Wilson’s disease
Alpha-1 anti-trypsin disease
Cirrhosis
Amebiasis
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Primary biliary cirrhosis
Secondary biliary cirrhosis
Primary sclerosing cholangitis
Wilson disease
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Pancreatic insufficiency
Iatrogenic
Infectious
Lymphatic obstruction
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Adenocarcinoma of the colon.
Large bowel obstruction.
Hemorrhagic ovarian cyst.
Ventral hernia.
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HCV is more likely than HBV to cause symptomatic hepatitis
HBV is more readily transmitted by sexual intercourse than is HCV
HBV is more likely than HCV to cause chronic life-long infection
HCV is more readily passed from mother to child at birth than is HBV
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Increased cholesterol secretion in the gall bladder
Increased pigment deposition in the gall bladder
Bacterial infection
Viral infection
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4
6
8
10
12
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Viral or bacterial infections
Furosemide or estrogens
Hyperlipidemia or hypercalcemia
Thrombosis and ischemia
Alcohol or biliary tract disease
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If virus is replicating in severe disease, HBV DNA will be positive and HBeAg will be negative.
After resolution of illness 2 months later, HBsAg remains positive and IgG antibody to HBsAg rises.
If she develops chronic hepatitis, HBsAg remains positive and IgG antibody to HBCAg is negative.
After onset of acute infection, IgM antibody to HBCAg rises before IgM antibody to HBsAg.
Cirrhosis
Acute hepatitis
Leptospirosis
Gallstones
Hepatocellular carcinoma
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He stands about an 80% chance of the condition becoming chronic
He will recover without any treatment
He stands about a 5% chance of the disease becoming chronic
You think he may be overdosing on acetaminophen and he is likely to continue this way unless you intervene
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Viral hepatitis
Gallstones
Hereditary spherocytosis
Dubin Johnson syndrome
Gilbert’s syndrome
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Mesenteric embolism
Ulcerative colitis
Crohn’s disease
Carcinoid syndrome
Peutz Jeghers syndrome
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Impaired hepatic blood inflow
Impaired intrahepatic blood flow
Impaired hepatic blood outflow
Portal vein obstruction
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Acute hepatitis
Chronic hepatitis
Liver cirrhosis
Hepatic steatosis
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A
B
C
D
E
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