Explore key pharmacological agents impacting cardiac function in this focused quiz. Assess your understanding of medications like Clonidine, Metoprolol, and Verapamil, and their roles in treating cardiovascular conditions. Essential for students and professionals in healthcare.
K+ channel blocker
Decreases AP duration
Antihypertensive
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Increase NaCl transport in the distal convoluted tubule
Used in patients with mild heart failure and edema
Useful in angina pectoris
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Na+ channel blocker
Causes increased excitation
Good antiarrhythmic
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Antihypertensive
Alpha 2 agonist
Causes increased NE and therefore decreased HR and decreased BP
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ACE inhibitor
Decreases contraction of smooth muscle
Used in patients with left ventricular dysfunction and no edema
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Cause decreased water absorption by inhibiting an ion cotransporter in the loop of Henle
Used for pulmonary edema and severe hypertension
Can be taken in combination with a sympatholitic and a vasodilator
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Calcium channel blocker
Causes vasodilation
Increased HR
Used to treat angina pectoris
Antihypertensive and antiarrhythmic
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Causes increased cGMP
Causes vasodilation
Used as an antiarrhythmic
Should be administered with sildenafil
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Inhibit Na+/K+ ATPase which increases Na+in the cell
Decreases intracellular Ca2+
Causes decreased SNS activity - decreases SNS barroreceptor response (won't get the vasodilation of blood vessels to compensate for increases HR)
Quinidine helps clear digoxin faster to avoid toxicity
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ACE inhibitor
Vasodilation
Bradykinin is the cause of vasodilation
Used in patients with L.ventricular failure and edema
Antihypertensive
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Decreases cGMP in smooth muscle which causes relaxation
Used in patients with edema
Is an antihypertensive
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Common name is Viagra
Inhibits phosphodiesterase
Causes increased cGMP
Contraction of smooth muscle
Causes tacchycardia when mixed with nitrites
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Alpha 1 blocker
Causes vasoconstriction and the barroreceptors then decrease HR
Increases renin release to increase blood volume
Administer in combination with a beta-blocker or a diuretic
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Non-selective beta blocker
Decreases HR which lowers BP
Decreases renin release from kidney (helps to decrease blood volume and lower BP)
Decreased HR means decreased myocardial O2 requirement = reason it's useful in angina pectoris
Antiarrhythmic
Antihypertensive
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Binds AT1 and AT2
Vasodilator
Binding AT1 presynaptically causes increased NE release
AT1 receptors on smooth muscle cause decreased Ca2+
Causes increased aldosterone and ADH which increased blood volume
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Opens K+ channels to cause hyperpolarization which results in vasodilation
Barroreceptor tries to increase HR
Is an antihypertensive
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