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Antihypertensive
Alpha 2 agonist
Causes increased NE and therefore decreased HR and decreased BP
Non-selective beta blocker
Decreases HR which lowers BP
Decreases renin release from kidney (helps to decrease blood volume and lower BP)
Decreased HR means decreased myocardial O2 requirement = reason it's useful in angina pectoris
Antiarrhythmic
Antihypertensive
Alpha 1 blocker
Causes vasoconstriction and the barroreceptors then decrease HR
Increases renin release to increase blood volume
Administer in combination with a beta-blocker or a diuretic
Opens K+ channels to cause hyperpolarization which results in vasodilation
Barroreceptor tries to increase HR
Is an antihypertensive
Decreases cGMP in smooth muscle which causes relaxation
Used in patients with edema
Is an antihypertensive
Calcium channel blocker
Causes vasodilation
Increased HR
Used to treat angina pectoris
Antihypertensive and antiarrhythmic
ACE inhibitor
Vasodilation
Bradykinin is the cause of vasodilation
Used in patients with L.ventricular failure and edema
Antihypertensive
ACE inhibitor
Decreases contraction of smooth muscle
Used in patients with left ventricular dysfunction and no edema
Cause decreased water absorption by inhibiting an ion cotransporter in the loop of Henle
Used for pulmonary edema and severe hypertension
Can be taken in combination with a sympatholitic and a vasodilator
Increase NaCl transport in the distal convoluted tubule
Used in patients with mild heart failure and edema
Useful in angina pectoris
Inhibit Na+/K+ ATPase which increases Na+in the cell
Decreases intracellular Ca2+
Causes decreased SNS activity - decreases SNS barroreceptor response (won't get the vasodilation of blood vessels to compensate for increases HR)
Quinidine helps clear digoxin faster to avoid toxicity
Causes increased cGMP
Causes vasodilation
Used as an antiarrhythmic
Should be administered with sildenafil
True
False
Common name is Viagra
Inhibits phosphodiesterase
Causes increased cGMP
Contraction of smooth muscle
Causes tacchycardia when mixed with nitrites
Na+ channel blocker
Causes increased excitation
Good antiarrhythmic
K+ channel blocker
Decreases AP duration
Antihypertensive
Binds AT1 and AT2
Vasodilator
Binding AT1 presynaptically causes increased NE release
AT1 receptors on smooth muscle cause decreased Ca2+
Causes increased aldosterone and ADH which increased blood volume
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