Hazards Of Supplemental Oxygen

I hate fill in the blank because it is impossible to get everybody to enter the same thing, and there is nothing more frustrating then getting a question wrong just because you used a capital letter.  Oh well, let's do it anyway, just follow the rules!  Oxygen toxicity is caused as a result of ____ ________. (2 words, 1st word 4 letters, 2nd 8 letters all lower case with a space inbetween)

Oxygen toxicity primarily affects the _____ and the ___.  (2 words, first word 5 letters, second "word" 3 letters, 1 space in between, and once again all lower case)

The two primary factors of O2 toxicity are PIO2 AND exposure time.

CNS effects only occur at pressures lower than 1ATM.

Damage to lung tissue can occur at clinical O2 levels.

What is the vasoconstrictive reaction that causes necrosis and proliferation of vessels leading to scar tissue, retinal detachment, and possible blindness in newborns called? (1 of it's names anyway, I've seen at least 3 so far)

Absorption atelectasis is a reaction to increased FIO2 that results in a decrease in:

Absorption atelectasis can occur in patients breathing room air.

According to the book (Egan's p.872) this is a natural response that prevents absorption atelectasis in alert individuals.

Major alveolar injury occurs after the exudative phase in pulmonary injury of hypoxia.

The "goal" of O2 therapy is to have the patient:

It is ALWAYS necessary to ween patients from 100% O2 within the 24 hour window.

Adults can usually breathe 50% FIO2 for extended periods of time without any damage.