Again, SOME questions from chapter 11 that WILL be on the exam. :D
Smooth and cardiac muscle
Skeletal muscle
Cardiac muscle
Smooth muscle
Skeletal, smooth and cardiac muscle
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In the liver
In the muscle tissue
In the small intestine
In the neuromuscular junction
In the sarcoplasmic reticulum
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9 ATP per second
7 ATP per second
5 ATP per second
1 ATP per second
3 ATP per second
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Isometric contraction
Isotonic contraction
Treppe
Tetanus
Isokinetic contraction
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Type I slow oxidative fibers
Type IIA intermediate glycoytic fibers
Type IIB fast glycolytic fibers
Type I slow-twitch fibers
Red fibers
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Red fibers
Intermediate fibers
Fast-twitch fibers
White fibers
Slow oxidative fibers
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Multiunit smooth muscle
The sarcoplasmic reticulum of skeletal muscle
The A bands of skeletal and cardiac muscle
Intercalated discs of cardiac muscle
The sarcolemma of skeletal muscle
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It does not have Z disc
It gets nearly all of its Ca2+ from the extracellular fluid
It does not have intercalated discs
It is regulated by a length-tension relationship
It is voluntary muscle
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None of them
contracts more slowly but relaxes more rapidly
Contracts more rapidly but relaxes more slowly
Contracts more slowly
Contracts and relaxes more slowly
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Protease
Myokinase
Cholinesterase
Acetylvholinesterase
Acid phosphatase
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The latent period
The threshold
Twitch
Recruitment
Innervation
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Fatigue
Treppe
Complete tetanus
Incomplete tetanus
Flaccid paralysis
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The muscle lengthens but tension remains constant
The muscle shortens but tension remains constant
The muscle tenses and shortens
The muscle tenses but length remains unchanged
The muscle lengthens and tension declines
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Intermediate fibers
Smooth muscle
Cardiac muscle
Fast glycolytic fibers
Slow oxidative fibers
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0
1
4
2
3
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Juctional folds
Terminal cisterna
Synaptic knob
Sarcolemma
Sarcoplasmic reticulum
Atrophy
Tetanus
Muscle wasting
Numbness
Flaccid paralysis
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To inhibit Ca2+ release from the sarcoplasmic reticulum
To inhibit the function of cholinesterase
To decrease the number of synaptic vesicles
To increase the number of ACh receptors
To promote multiple motor unit summation
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Treppe
Wave summation
Recruitment
Incomplete tetanus
Complete tetanus
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24
48
12
36
100
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Longer aerobic respiration
Increased glycolysis
Increased use of myokinase
Longer anaerobic fermentation
Reduced ATP consumption
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Endomysium
Sarcomeres
Satellite cells
Dense bodies
Somatic nerve fibers
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Deep respiration
Goose bumps
Blood leaving the left ventricle of the heart
Blinking of the eyelids
Elevating the eyebrows
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Two thin myofilaments and one thick myofilament
Two terminal cisternae and one T tubule
To thick myofilaments and one thin myofilament
Two A bands and one I band
Two T tubules and one terminal cisternae
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Recruit more muscle fibers
Lower threshold
Reduce its wave summation
Shift from isometric to isotonic contraction
Shift from slow-twitch to fast-twitch mde
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F actin
Tropomyosin
Troponin
Titin
Dystrophin
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Prevent calcium from entering cardiac muscle, thus slowing down the heart rate.
Prevent calcium from exiting the sarcoplasmic reticulum in skeletal muscle
Prevent calcium from entering the sarcoplasmic reticulum of the smooth muscle.
Prevent calcium from entering smooth muscle, thuse allowing the muscle to relax.
Stimulate the calcium pump in smooth muscle, thus removing calcium from the calmodulin
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No effect
Myocardial atrophy
Lockjaw
Spastic paralysis
Flaccid paralysis
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Between the thin myofilaments and the sarcolemma of a muscle fiber
As a transmembrane protein of the sarcolemma
In the Z discs of skeletal and cardiac muscle
In the dense bodies of smooth muscle
Bridging the gap between the sarcolemma and synaptic knob
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ATP is regenerated faster than it is consumed
Myosin heads show faster and faster power strokes
Ca2+ accumulates in the sarcoplasm faster than the sarcoplasmic reticulum can reabsorb it.
More and more ACh is released with each stimulus
As the muscle warms up, aerobic respiration is accelerated
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To prevent single-unit smooth muscle cells from pulling apart
To release norepinephrine
To reabsorb the decomposition products of ACh after AChE breaks it down
To link the thin filaments to the inside of the sarcolemma in smooth muscle
To enable each cardiac myocyte to directly stimulate its neighbors
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Donates one of its phosphate groups to ADP
Phosphorylates and activates certain enzymes in the sarcoplasm
Catalyze the transfer of phosphate from CP to ADP
Functions as a substitute for ATP during anaerobic fermentation
Acts as a second messenger in muscle fibers.
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Increases permability of the sarcolemma
Lowers the threshold of the muscle fiber
Overrides the inhibitory effect of AChE
Decreases the positive charge on the sarcolemma
Decreases the release of Ca2+ from the sarcolemma
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The intracellular environment has more positively charged sodium
It has a voltage of about +75 mV
It depends on the muscle fiber absorbing potassium ions fro the ECF
The intracellular environment is negatively charged
The extracellular environment is negatively charged
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Potassium
Calcium
Sodium
Magnesium
Lithium
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The triad removes ACh from the synaptic cleft
The triad allows for a Ca2+ release when a muscle fiber is excites
The triad maintains the resting membrane potential
The triad synthesizes ATP
The triad stores sodium
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The respiratory system
The glycolysis system
The anaerobic system
Phosphagen system
Glycogen-lactic acid system
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Myopathy
Dystrophy
Atrophy
Apathy
Treppe
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Plasticity
Contractility
Conductivity
Extensibility
Excitability
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Excitable
Impermeable
Elastic
Expendable
Contractile
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A myofibril
A thin filament
A myoglobin
An elastic filament
a sarcoplasm
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Longevity
Plasticity
Tone
Tensile strength
Elasticity
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Fully relaxed before being stimulated.
In a state of treppe.
Well-rested and low in creatine phosphate.
Partially stretched before being stimulated.
Greatly stretched before being stimulated.
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have a smaller mitochondria
Have more glycogen in them
Make more use of aerobic respiration
Do not have as many blood capillaries per gram of tissue
Break ATP down to ADP and Pi faster
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Tropomyosin
F actin
Dystrophin
G actin
Myosin
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Treppe
Striations
Myofibrils
Strabisums
Myoblasts
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Convert certain parallel muscles into stronger pennate muscles
Lower the threshold for muscle excitation
Increase the size of his motor units
Increase the average number of myofibrils per muscle fiber
Convert white muscle tissue to red muscle
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