Na+-K+ ATPase pumps are not active
The resting membrane potential is about +65 mV
K+ ions leak out of a neuron via nongated K+ channels
Voltage-gated K+ channels are open
The resting state represents the relative refractory period
Receptors for gamma aminobutylic acid (GABA)
Nongated Na+ channels
Voltage-gated Ca++ channels
Voltage-gated Na+ channels
Release of neurotransmitter Ach
Form choroid plexus that produce cerebrospinal fluid
Transport glucose from capillaries to neurons
Give rise to perivascular end feet in the dorsal root ganglion
Stop dividing shortly after birth
Promote intercellular accumulation of neurotransmitter glutamate
Central canal
Neuronal perikarya
Ependymal cells
Schwann cells
Oligodendrocytes
An excitatory postsynaptic potential only
An inhibitory poststynaptic potential only
No obvious overall changes in membrane potential
Hyperpolarizing potential
An action potential
Presence of quadriceps reflex
Absence of quadriceps reflex
Slight increase in conduction velocity of action potential
Decreased conduction velocity to the speed typically seen in nonmyelinated nerve fibers
A loss of conduction of action potentials as local currents cannot reach the voltage-gated Na+ and K+ channels
Increased conduction velocity due to a loss of resistance imposed by axonal plasma membrane
No apparent change in conduction speed as voltage-gated Na+ and K+ channels are still available in the demyelinated area
Sensory neurons in the dorsal horn
Postsynaptic acetycholine receptors
Release of Ach at the neuromuscular junction
Release of glycine from the pre-synaptic site
Motor neurons in the ventral horn
Nerve fibers that form the neuromuscular synapse leave the spinal cord via the dorsal root
Terminal branches of the motor end plate release glutamate
Neuromuscular synapse is affected by myasthenia gravis
Postsynaptic membrane has receptors for gamma aminobutylic acid (GABA)
Axon terminals are covered with astrocytes
Inhibitory postsynaptic potentials
Na+-K+ ATPase pumps
Opening of voltage-gated K+ channels
Inactivation of voltage-gated Na+ channels
Activation of voltage-gated Na+ channels
Glycine
Ach
GABA
All of the above
Made of an axon and endoneurium
Made of an axon and Schwann cells
Made of a myelinated axon and endoneurium
Made of a nerve fasicle surrounded by perineurium
Made of an axon and oligodendrocytes
Ligand-gated receptors to trigger influx of Cl-
Ligand-gated receptors to trigger influx of Na+
Voltage-gated Na+ channels to trigger influx of Na+
Voltage-gated K+ channels to trigger influx of K+
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