Review of material for test one going over pain pathways and pain management.
Unmyelinated, myelinated
Slow, Fast
Myelinated, unmyelinated
Thin, thick
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First Order neurons
Second Order Neurons
Third Order Neurons
All The Above
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Bradykinin
Serotonin
Histamine
Epinephrine
First order Neurons
Second order Neurons
Third order Neurons
All the Above
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PostCentral Gyrus
Limbic System
Amygdala
Medulla
First order neurons - from peripheral tissue to dorsal horn
Second order neurons - from dorsal horn to thalamus
Third order neurons - from thalamus to post central gyrus
All of the above are correct
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A delta fibers are fast, sharp, myelinated, and well-localized
All A fibers (alpha, beta, gamma, and delta) are myelinated
C fibers are non-myelinated, slow, and poorly localized.
A fibers are responsible for throbbing, aching postop pain
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First Order Neurons
Second Order Neurons
Third Order Neurons
All The Above
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Radiculopathy
Anesthesia Dolorosa
Allodynia
Hyperalgesia
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Heat damage
Mechanical damage
Disease damage
Chemical damage
Acute Deep Somatic
Acute True Visceral pain
Acute true Parietal pain
Acute Superficial somatic pain
Are carried by facial nerves to the brain stem where they synapse with second order neurons.
Synapse with second order neurons in the dorsal horn
Are much thicker and transmit non painful stimuli
None of above
Surgical incision into the kidney
A surgical incision into the skin
Both are equally painful
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Lamina I
Lamina V
Lamina IV
Lamina II
Lipoxygenase
Factor XII
Phospholipase A
Cyclooxygenase
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Mechanonociceptor
Silent Nociceptor
Polymodal mechoheat nociceptors
Somatic Nociceptors
Wow, she actually though that was english?
Ken Leeeeeeeee!!!!!
Stupid pixar removed their cartoons from you tube.....
All the above
Mu receptors
Lamina 2 receptors
NK-1 receptors
P45 receptors
A alpha fibers
C Fibers
A delta fibers
D fiber
Hypoalgesia
Analgesia
Parasthesia
Anesthesia
Histamine
Serotonin
Prostaglandins
Norepinephrine
Acute Deep Somatic Pain
Acute Visceral Pain
Chronic Pain
Acute Superficial Somatic Pain
Prostaglandin E
Cyclo-oxygenase
Phospholipase A
Lipoxygenase
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Leukotrienes
Prostacyclins
Prostaglandin E
Bradykinin
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Mechanonociceptor
Silent Nociceptor
Polymodal mechoheat nociceptors
Somatic Nociceptors
Functional abnormality of one or more nerve roots
Abnormal sensation perceived without an apparent stimulus
Pain in the distribution of a nerve or a group of nerves.
Pain in an area the lacks sensation
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Pain that is the result of injury or acquired abnormalities of peripheral or central neural structures.
Pain that results directly from a disease process.
Pain causes by activation or sensitization of peripheral nociceptors, which transmit noxious stimuli.
None of the above
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Histamine released from mast cells
Phospholipids released from damaged cell membranes
Release of alogens from damaged cells
Bradykinin released from tissues
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Glutamate
Substance P
Dynorphin
Capsaicin
Functional abnormality of one or more nerve roots
Abnormal sensation perceived without an apparent stimulus
Pain in the distribution of a nerve or a group of nerves.
Pain in an area the lacks sensation
Decreased minute ventilation and CO2 production
HTN and increased myocardial irritability
Decreased intestinal and urinary motility
Hypercoagulability
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Mechanonociceptor
Silent Nociceptor
Polymodal mechoheat nociceptors
Somatic Nociceptors
Glutamate
Substance P
Dynorphin
Capsaicin
Hyperalgesia
Dysesthesia
Anesthesia Dolorosa
Radiculopathy
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Radiculopathy
Dysesthesia
Hypalgesia
Parasthesia
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Hyperpathia
Radiculopathy
Hyperneuralgia
Acute Polyradiculopathy
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Functional abnormality of one or more nerve roots
Abnormal sensation perceived without an apparent stimulus
Pain in the distribution of a nerve or a group of nerves.
The sensation I am feeling right now having to study all the BS.
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Pain that is the result of injury or acquired abnormalities of peripheral or central neural structures.
Pain that results directly from a disease process.
Pain caused by activation or sensitization of peripheral nociceptors, which transmit noxious stimuli.
None of the above
First Order Neurons
Second order neurons
Third order Neurons
All the Above
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Is a COX inhibitor, so it prevents arachidonic acid from converting to Prostaglandins which causes pain.
Blocks activation of phospholipase A to inhibit prostanglandin production.
Is a leukotriene inhibitor and breaks down bradykinin.
Inhibits transmission of Substance P by blocking NK-1 receptors.
C fibers
B fibers
A fibers
All the above
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Parasthesia
Allodynia
Hyperalgesia
Neuralgia
Anesthesia Dolorosa
Allodynia
Hypesthesia
Parasthesia
Through inhibition of the COX pathway, which turns arachidonic acid into PGE.
Through inhibition of phospholipase A which acts on phospolipds to make arachidonic acid
Through inhibition of bradykinin which relieves inflammation
All the above
Location, density and duration of pain.
Arousal and autonomic responses to pain.
Unpleasant emotional perception of pain.
Activates antinociceptive descending pathways.
Lamina II, III, V
Lamina V only
Lamina I, V
Lamina I, II, III
Wind up and sensitization of second order neurons
Receptor field expansion
Up regulation of adrenergic receptors
Hyperexcitability of flexion reflexes
True
False
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