Questions from pre-lab quiz and post-lab quiz about hormone replacement therapy.
Is produced by developing ovarian follicles.
Is a peptide hormone.
Is an anterior pituitary hormone secretion.
Inhibits bone growth and, therefore, protects against the development of osteoporosis.
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The ovaries stop secreting estrogen after menopause.
An untreated female's T score in anticipated to become more positive after menopause.
Surgical removal of the ovaries will help limit the potential health problems associated with menopause.
Osteoporosis is an increase in the density of porous long bones.
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A placebo on bone density.
Large needles on the behavior of rats.
Potassium chloride on bone density.
Sodium chloride on bone density.
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Prevent goiter formation in this rat through its effect on negative feedback to the thyroid gland.
Make the rat crave more milk and other calcium-rich beverages.
Have no effect on bone density and, therefore, should be excluded from rat hormone-replacement therapies.
Inhibit osteoclast activity and stimulate calcium uptake and deposition in long bones.
Stimulates osteoclast activity and inhibits calcium uptake into long bones.
Is an anterior pituitary hormone secretion.
Is a steroid hormone.
Works against the development of osteoporosis.
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The X-ray scanning assay reports a more-negative T-score
The rat requires a higher dose of hormones to be injected for adequate therapy.
The X-ray scanning assay reports a less-negative T-score.
The rat requires more gaseous anesthetic to remain immobilized.
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Estrogen levels were reduced because of the increased negative feedback caused by the injections.
Estrogen levels were elevated because of the loss of negative feedback control.
Osteoporosis was evident prior to the injections of estrogen.
Follicle-stimulating hormone (FSH) levels were reduced because of the loss of negative feedback control.
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