A quiz reviewing the material for Quiz 3. Sympathomimetics, Calcium channel blockers, Peripheral Vasodilators.
Phenylephrine
Norepinephrine
Clonodine
Dopamine
Ha ha!! Now that is how you handle a telemarketer.
Poor telemarketer, he sounds like he is gonna piss his pants.
Evil.... but i like it!
All the Above
By antagonizing adrenergic receptors
Through blockade of Na channels.
By blocking the slow inward calcium driven current
By prolonging repolarization
Digitalis Toxicity
Lidocaine Toxicity
Amiodarone Toxicity
Phenytoin Toxicity
ST elevation on ECG
Low serum K
ABG showing hypoxia
Diabetes
Beta 2
Alpha 1
Beta 1
Alpha 2
Alpha 1
Alpha 2
Beta 1
Beta 2
Dysrhythmias
Renal Failure
COPD
Hypertensive ischemic heart disease
Maintaining respiratory stability
Sedation
Reduces brain Noradrenergic Activity
Increase pt BP
Decrease SVR
Vasoconstriction
Increase LV afterload
Increase arterial BP
Beta 2 agonist
Blocks calcium channels of AV node
Directly relaxes arterial smooth muscle
Imitates cGMP in smooth vasculature
Re-uptake
Diffusion from receptor sites
Irreversible protein binding
Enzyme degredation
Vanillymandelic Acid
3,7-dicarboxyvandelic acid
Nelson-Mandelic Acid
4,6-Pheocomtic acid
By antagonizing adrenergic receptors
Through blockade of Na channels.
By blocking the slow inward calcium driven current
By prolonging repolarization
Nifedipine
Diltiazem
Isoproteronol
Nimodipine
Hypokalemia
Hypochloremia
Hyponatremia
None of above
4.5 mg/kg
10 mg/kg
2 mg/kg
5 mg/kg
Synaptic Vessicles
Mitochondria
Golgi Bodies
Sarcoplasmic Reticulum
Epinephrine
Amphetamine
Dopamine
Ephedrine
Dopamine
Norepinephrine
Epinephrine
Acetylcholine
Phenylephrine
Dopamine
Dobutamine
Norepinephrine
Repolarization to resting potential results from outward K current
Rapid phase of repolarization caused by inactivation of Na influx and activation of a transient outward K current
Voltage dependent Na channel opens and rapid sodium influx depolarizes cell
Plateau phase, characterized by low membrane conductance and the activation of slow inward Ca current
Outward K current is deactivated and an inward Na current reduces transmembrane potential
Opening of an ion channel
Activation of a second messenger such as cAMP or inositol phosphate
Depolarization
All of above.
Increase MAC
Decrease MAC
No effect on MAC
Beta 2 adrenoreceptor agonist
Alpha 2 adrenoreceptor agonist
Potentiation of GABA
None of above
Nifedipine
Nicardipine
Nimodipine
Hydralyzine
Increase Contractility
Increase HR
Increase CO
Decreased SVR
Phenylephrine
Amphetamine
Isoproterenol
Dobutamine
N-methyltransferase
Dopamine B-Hydroxylase
DOPA decarboxylase
Dopamine does not directly turn into norepi… you’re missing a step.
Acetylcholine
Norepinephrine
Epinephrine
Tyrosine
By antagonizing adrenergic receptors
Through blockade of Na channels.
By blocking the slow inward calcium driven current
By prolonging repolarization
By antagonizing adrenergic receptors
Through blockade of Na channels.
By blocking the slow inward calcium driven current
By prolonging repolarization
Calcium, Magnesium
Potassium, Sodium
Magnesium, Calcium
Sodium, Potassium
Norepinephrine
Amphetamine
Ephedrine
Phenylephrine
Post-synaptic Neurons
Pineal Body
Posterior Lobe of Pituitary
Synaptic Vessicles
Norepinephrine
Ephedrine
Phenylephrine
Epinephrine
Altered sympathetic nervous system activity
Venous Hypoxia
Myocardial Ischemia
Bradycardia
Dopa
Phenylalanine
Norepinephrine
Epinephrine
Phenytoin
Amiodarone
Procainamide
Lidocaine
Watch your Arterial BP very closely, these drugs together cause profound HTN and increase risk for stroke
Monitor pt for cyanide toxicity
Send serum K levels and watch for arrythmias
Monitor for hpoxemia and seizure activity
Acetylcholine
Norepinephrine
Epinephrine
Tyrosine
Isoproterenol
Phenylephrine
Ephedrine
Calcium Chloride
Vifedipine
Epinephrine
Dopamine
Phenylephrine
10
5
100
20
They are not metabolized by MAO or COMT
Higher degree of protein binding
Increase volume of distribution
All of above
Nifedipine
Verapimil
Nimodipine
Diltiazem
Phenytoin
Digoxin
Lidocaine
Amiodarone
Calcium channel blockers prolong both depolarizing and non-depolarizing agents
Succinylcholine
Rocuronium
Cisatracurium
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