This quiz explores the immune system's role in oral health, focusing on how it responds to antigens, memory functions, and key immune cells involved in oral pathology.
Produce antibody called IgE
React with lymphocytes
Combine antigen
Form immune complexes
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B cell
Plasma cell
T cell
Macrophage
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Increasing the risk of an antigen causing disease
Using antibodies produced by another person
Passing antibodies from the mother to the fetus
Producing active acquired immunity
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Phagocytosis
Margination
Titer
Pavementing
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Chronic inflamatory
Oportunistic
Hyperplastic
Granulomatous
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Bacterium
Virus
Fungus
Protozoan
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Also termed immunoglobins
Directly produced from lymphocytes
Produced in response to other antibodies
Directly produced from mast cells
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Pemphigus
Erosive lichen planus
Desquamative gingivitis
Lupus erythematosus
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Leukoplakia
Geographic tongue
Erthema multiforme
Xerostomia
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Antigens
Antibodies
Autoimmune cells
Toxins
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Hypersensitivity
Immunodeficiency
Hyperplasia
Autoimmune disease
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Lichen planus
Urticaria
Angioedema
Contact mucositosis
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Cicatrical pemphigoid
Pemphigus vulgaris
Lichen planus
All of the above
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Plasma cells
Antibodies
Antigens
Lymphocytes
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It retains memory of the antibody
It allows faster future immune responses
Like inflammation it remembers the antigen
It weakens future immune responses
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An infectious disorder
An automatic response
An immunologic disorder
More common in women than in men
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Pemphigus vulgaris
Erythema multiforme
Systemic lupus erythematosus
Behcet's syndrome
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Behcet syndrome
Histocytosis X
Ulcerative colitis
Crohn's disease
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Lips
MUCOSA
EYELIDS
Epiglottis
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Plaque-like
Erosive
Papular
Reticular
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Behcet syndrome
Systemic lupus erythematosus
Lichen planus
Erythema multiforme
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The primary lesion of syphillis is called a chancre
The secondary lesion of syphillis occurs at the site of inoculationwith the organism
The tertiary lesion of syphillis is called a gumma
Syphillis is caused by the spirochete Treponema pallidum
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Hand-schuller-christian disease
Eosinophilic granuloma
Letterer-siwe disease
Chronic disseminated reticulosis
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Type 1
Type 2
Type 3
Type 4
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Size of the ulcerations
Age and sex of the patient
The histologic findings
Amount of keratosis seen in both
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Angular chelitis may be caused by candid albicans
White lesion resulting from candiasis may not rub off
Erythematous candidiasis is usually completely asymptomatic
Denture stomatitis may be a form or oral candidiasis
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Natural passive immunity
Acquired passive immunity
Natural active immunity
Acquired active immunity
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Ulcer formation
Pain
Unilateral distribution of lesions
White lesions
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Nicotine stomatitis
Lichen planus
Angioedema
Geographic tongue
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Letterer-siwe disease
Hand-schuller-christian disease
Eosinophilic granuloma
Behcet's syndrome
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Hairy leukoplakia
Herpangina
Nasopharyngeal carcinoma
Infectious mononucleosis
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NK cells do not circulate
NK cells secrete antibodies
NK cells can recognize antigen
NK cells do not have memory
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Lymphocytes and plasma cells
Fibroblasts and lymphocystes
Eosinophils and mononuclear cells
Neutrophils and lymphocytes
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Osteoblasts
Fibroblasts
B lymphocytes
Erythrocytes
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Clinically resembles an irritativ efibroma
Is caused by a human papilloma virus
Is most commonly seen on the buccal mucosa
All of the above
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The bullae of pemphigus culgaris are more fragile than those o bullouw pemphigoid
Acantholysis of the epithelium is seen in pemphigus vulgaris
In pemphigoid the separation of the epithelium from the connective tissue occurs in the area of the basement membrane
Skin lesions are common in cicatrical pemphigoid
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Retains the memory of the encountered antigen
Serves as a link between the inflammatory and repair process
Undergoes B cell phagocytosis initially during inflammation
Can be activated by lymphokines
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Coxsackie virus infection
Human papillomavirus infection
Tuberculosis and syphillis
Candidiasis and herpessimplex infection
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