Skin turgor.
Temperature.
Thirst.
Daily weight.
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Anorexia.
Dizziness.
Rapid weight gain.
Poor skin turgor.
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Maintenance of medication compliance.
Avoidance of normal activities with stress.
Adherence to a 2-g sodium diet.
Prevention of hypertensive episodes.
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Returning to work after a weekend.
Going on vacation.
Having oral surgery.
Having a routine medical checkup.
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Hypersensitivity to sun exposure.
Increased serum bilirubin level.
Adverse effects of the glucocorticoid therapy.
Increased secretion of adrenocorticotropic hormone (ACTH).
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Orthostatic hypotension.
Muscle hypertrophy in the extremities.
Bruised areas on the skin.
Decreased body hair.
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Weight loss.
Thin, fragile skin.
Hypotension.
Abdominal pain.
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Glucocorticoids and aldosterone.
Adrenocorticotropic hormone (ACTH).
Glucocorticoids, aldosterone, and androgens.
Catecholamines.
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Postprandial hypoglycemia.
Hypokalemia.
Hyponatremia.
Decreased urine calcium level.
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Some clients are very sensitive to the effects of cortisol and develop symptoms even with normal levels.
A single random blood test cannot provide reliable information about endocrine levels.
The excessive cortisol levels seen in Cushing’s disease commonly result from loss of the normal diurnal secretion pattern.
Tumors tend to secrete hormones irregularly, and the hormones are generally not present in the blood.
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Increase calories.
Restrict sodium.
Restrict potassium.
Reduce fat to 10%.
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Increase the amount of potassium in the diet.
Maintain a regular program of weight-bearing exercise.
Limit dietary vitamin D intake.
Perform isometric exercises.
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“Sit in an upright position and take a deep breath.”
“Hold your abdomen firmly with a pillow and take several deep breaths.”
“Tighten your stomach muscles as you inhale and breathe normally.”
“Raise your shoulders to expand your chest.”
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Beginning oral nutrition.
Promoting self-care activities.
Preventing adrenal crisis.
Ambulating in the hallway.
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