IgE is involved in both anaphylactic and immune complex hypersensitivibes
Complement is involved in both anaphylactic and immune complex hypersensitivities
Less antigen is typically needed to trigger and anaphylactic reaction than an immune complex reaction
Neutrophils play more important role in anaphylactic reactions than in immune complex reactions
Type I Hypersensitivity
Type II Hypersensitivity
Type III Hypersensitivity
Type IV Hypersensitivity
Direct activation of mast cells by the anesthetic gas
Type I
Type IV
Type II
Type II or III
Type III
The class (isotype of antibody)
Whether the antibody reacts with the antigen on the cell or reacts with antigen before it interacts with the cell
The participation of complement
The participation of T cells
IgE antibody
IgG antibody
Sensitized T cells
Complement
IgM antibody
IL-2
Leukotrienes
Serotonin
Bradykinin
Mediated by IgE antibody
Mediated by IgG and IgM antibody
Intiated by haptens
Initiated by Th2 cells
IgG and complement
IgE and histamine
IL-2 and cytotoxic T cells
Gamma interferon and macrophages
Systemic anaphylaxis
Serum sickness
An Arthus reaction
Cytotoxic hypersensitivity
Edema without a cellular infiltrate
An infiltrate composed of neutrophiis
An infiltrate composed of helper T cells and macrophages
An infiltrate composed of eosinophils
Bacteria that adhere to vascular walls inducing necrosis
IgM immune complexes that induce vasculitis
Immune-mediated destruction of bacteria, releasing endotoxin
The disease is a the result of superantigen activity by the infectious agent
IgA immune complexes that induce vasculitis
Type I Hypersensitivity reaction
Type II Hypersensitivity reaction
Type IV Hypersensitivity reaction
Anaphylactoid reaction
Type III Hypersensitivity reaction
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