Nsaids - Part 1 - Cox Enzyme Functions (Msq Drill 177)

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1. Eventually the prostaglandins form different tissue isomerase that play multiple roles in various tissues in the body. True or false?
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2. Patients on NSAIDS are at risk for analagesic nephropathy. True or false?
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3. Are prostaglandins also produced from arachidonic acid?
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4. If one therefore blocks COX 1, the gastric mucosa will:
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5. If you give a patient a COX inhibitor with bother COX 1 and 2 effect, this will slow down or eliminate macrophage taxis. Will this lead to increased or decreased inflammation?
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6. Therefore a patient can develop Acute renal failure due to NSAIDS. True or false?
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7. COX 1 initiates the process that eventually leads to the production of ___________  which is responsible for platelet aggregation.
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8. Decreased renal afferent flow will lead to:
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9. Taking an NSAID will reduce perfusion into the kidneys. The kidneys respond by raising or lowering blood pressure?
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10. Arachidonic acid is released from ______ .

Explanation

Our cell membrane plays a role in the production of arachidonic acid.

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11. Since prostaglandins can be manipulated by COX 1 and COX 2 to form various tissue isomerases. Which COX enzyme is responsible for maintaining the mucosa, facilitating cytoprotective secretions and increasing secretions?
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12. If all I wanted was an antipyretic effect from an NSAID then I would choose:

Explanation

antipyretics - drugs that reduce fever

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13. If you increased the dose of the COX inhibitor will the inflammation be even more reduced?
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14. Which COX enzyme increases fever and also increases my sense of pain?

Explanation

COX 2 increased fever and also increases nocicpetion.

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15. Do COX enzymes cause vasodilation or vasoconstriction?
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16. Are leukotrienes produced directly from phospholipids or from arachidonic acid?
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17. So, Aspirin has an effect on COX 1 or COX 2?
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18. If you block COX enzymes by taking NSAIDs you increase or decrease leukotrienne formation?
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19. What do NSAIDS cause, an increase or decrease in pottasium?
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20. Do both COX enzymes affect microphage chemotaxis?
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21. One COX enzyme causes platelet aggregation whilst the other inhibits platelet aggregation. Which COX enzyme inhibits platelet aggregation?
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22. In general, COX enzymes perform the following functions on the kidney:
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23. If you give a patient an NSAID, they will excrete more or less sodium?

Explanation

COX enzyme generally facilitate sodium excretion and potassium retention.

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24. If COX enzymes cause vasodilation, what happens to renal afferent flow when a patient takes an NSAID?
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25. A physician is considering administering an NSAID to a woman in labor. Will this lead to a quicker delivery or an inhibition of the labor?
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26. What enzyme facilitates the release of arachidonoc acid from phospholipase?
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27. What product of arachidonic acid plays a role in pulmonary inflammation and bronchconstriction and therefore has a role in the asthma?
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28. Eventually patients on NSAIDs will have lower potassium levela and renal tubular _____ .
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29. COX 2 facilitates the production of ________ which is responsible for inhibiting platelet aggregation.
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30. Which COX enzyme causes vasodilation and thus increased perfusion to the kidneys?
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31. What eznymes facilitates the prduction prostaglandins from arachidonic acid?
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32. When a patient has an ulcer, their body probably uses COX _____ in the process of wound healing.
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33. What classes of antihypertensive medication should not be used concurrently with NSAIDS 
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34. Thus, increased leukotrienne formation will result in the following symptoms.

Explanation

anaphylactoid - resembling anaphylaxix

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35.
We have established that COX 2 causes platelet aggregation or thrombosis. IF you gave a patient Aspirin, will this thrombotic risk be decreased?
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Eventually the prostaglandins form different tissue isomerase that...
Patients on NSAIDS are at risk for analagesic nephropathy. True or...
Are prostaglandins also produced from arachidonic acid?
If one therefore blocks COX 1, the gastric mucosa will:
If you give a patient a COX inhibitor with bother COX 1 and 2 effect,...
Therefore a patient can develop Acute renal failure due to NSAIDS....
COX 1 initiates the process that eventually leads to the production of...
Decreased renal afferent flow will lead to:
Taking an NSAID will reduce perfusion into the kidneys. The kidneys...
Arachidonic acid is released from ______ .
Since prostaglandins can be manipulated by COX 1 and COX 2 to form...
If all I wanted was an antipyretic effect from an NSAID then I would...
If you increased the dose of the COX inhibitor will the inflammation...
Which COX enzyme increases fever and also increases my sense of pain?
Do COX enzymes cause vasodilation or vasoconstriction?
Are leukotrienes produced directly from phospholipids or from...
So, Aspirin has an effect on COX 1 or COX 2?
If you block COX enzymes by taking NSAIDs you increase or decrease...
What do NSAIDS cause, an increase or decrease in pottasium?
Do both COX enzymes affect microphage chemotaxis?
One COX enzyme causes platelet aggregation whilst the other inhibits...
In general, COX enzymes perform the following functions on the kidney:
If you give a patient an NSAID, they will excrete more or less sodium?
If COX enzymes cause vasodilation, what happens to renal afferent flow...
A physician is considering administering an NSAID to a woman in labor....
What enzyme facilitates the release of arachidonoc acid from...
What product of arachidonic acid plays a role in pulmonary...
Eventually patients on NSAIDs will have lower potassium levela and...
COX 2 facilitates the production of ________ which is responsible for...
Which COX enzyme causes vasodilation and thus increased perfusion to...
What eznymes facilitates the prduction prostaglandins from arachidonic...
When a patient has an ulcer, their body probably uses COX _____ in the...
What classes of antihypertensive medication should not be used...
Thus, increased leukotrienne formation will result in the following...
We have established that COX 2 causes platelet aggregation or...
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