Ventricular tachycardia; stroke; atherosclerosis
End organ damage; pulmonary oedema; thrombosis
Target organ damage; transient ischemic attack; retinopathy
Decrease cardiac output; decrease total peripheral resistance
Decrease cardiac output; decrease total blood volume
Decrease total peripheral resistance; decrease total blood volume
Decrease CO, decrease renin release, decrease sympathetic activity; propranolol, atenolol
Decrease CO, decrease renin release, increase sympathetic activity; salmeterol, salbutamol
Decrease CO, increase aldosterone release, increase parasympathetic activity; propranolol; salmeterol
Mimic ACE, preventing angiotensin 1 conversion to angiotensin 2; captopril (mimics AT2)
Inhibit ACE, preventing angiotensin 1 conversion to angiotensin 2; captopril (mimics AT1)
Inhibit renin, preventing angiotensinogen converstion to angiotensin 1; verapamil (inhibits ACE)
Block channel, stopping Ca entry assc with depolarisation; dihydropyridines e.g. verapamil are peripheral vasodilators; non-dihydropyridines e.g. nifedipine are equipotent for cardiac muscle and vasculature, reduce ionotropism (force of muscle contraction)
Block channel, stopping Ca entry assc with repolarisation; dihydropyridines e.g. niedipine are peripheral vasodilators; non-dihydropyridines e.g. verapamil are equipotent for cardiac muscle and vasculature, increase ionotropism (force of muscle contraction)
Block channel, stopping Ca entry assc with depolarisation; dihydropyridines e.g. verapamil are peripheral vasoconstrictors; non-dihydropyridines e.g. nifedipine are equipotent for cardiac muscle and vasculature, reduce ionotropism (force of muscle contraction)
Antagonism at postsynaptic alpha adrenoceptors, preventing Adr, preventing vasoconstriction; verapamil
Antagonism at postsynaptic alpha adrenoceptors, preventing NAdr, preventing vasoconstriction; prazocin
Antagonism at presynaptic alpha adrenoceptors, preventing Adr, preventing vasodilation; prazocin
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