This is before the decussation so the contralateral side will be affected.
Explanation
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C5 is below the pyramidal deccusation.
This is because the cortex inhibits the reflex. When you lose the cortex, you lose that inhibition and thus the hyperreflexia.
The white matter has tracts or axons.
No reflexes will be present. The LMN is responsible for the reflex arc.
AKA primary motor cortex
There is no corticol input. This is an involuntary, unconscious input from the sensory nerve into the spinal cord and straight back out to the muscle. There is no cortical input.
It has both sensory and motor neurons.
Lower motor neuron damage is synonymous with flaccid paralysis. Upper motor neuron damage will have a positive babinski.
This applies to lesions in the cortex as well. It is important to check where the lesion is. If the lesion is on the lateral cortex, the patient will still have no babinski because no damage has occured at the lower extremity area.
Upper and lower motor neurons.
These are the pyramids. So the cell bodies are in the ipsilateral motor cortex because the decussation is below this level.
The only damage at C5 would be LMN only. Below the lesion it will be total upper motor neuron damage. Reflexes would be heightened below the lesion.
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