The primitive heart tube undergoes a ventrocaudal folding down and to the left to ultimately to lie in its final resting position.
The right atrium consists of the right vitelline vein, primitive right atrium, and the sinus venarum.
The majority of the heart is derived from surrounding mesenchyme of endodermal original.
The flexible septum secundum functions as a primitive valve allowing for right to left shunting of blood in the fetus
A portion of the bulbis cordis (conus cordu) contributes to the aorticopulmonary septum
The right sinus venosus is obliterated and does not significantly contribute to the adult heart.
Ejection fraction is approx 55% and is normal, they have nothing to worry about
Ejection fraction is 40% indicating they may be experiencing some form of heart failure
Ejection fraction is 65% indicating they are under the influence of some positive inotropic agents
Ejection fraction is approx 40% and they have nothing to be concerned with, this is normal
Ejection fraction is approximately 55%, and they should seek consultation
Ejection fraction is 65% which is within the normal range
It is associated with the S4 heart sound
It is due to a prolonged increase in afterload
It is the result prolonged volume overload within the left ventricle
It is the sound made in response to the rebounding of pressure in the left atrium
Dilation of the ventricles
Left subclavian artery
Arch of Aorta
Left pulmonary artery
Commoon carotid artery
Small intestines to blood to liver to adipose cells via CII and LPL plasma interactions
Small intestines to lymph to blood to adipose cells via CII and LPL plasma interactions
Small intestines to lymph to blood to adipose cells via ApoB48 and LPL interactions
Small intestines to liver to blood to adipose cells via CII and LPL interactions.
Small intestines to lymphto liver via apoE to form LDL.
Abetaliportenemia and increased plasma triglycerides
Type IV dyslipidemia and tendon xanthomas
Type IIa dyslipidemia and xanthelasmas
Type IIa dyslipidemia and eruptive xanthomas
Type IV dyslipidemia and eruptive xanthomas
Increased stroke volume with overall increase of TPR
Increased cardiac output with decrease in pulse pressure
Increased Sympathetic Vasoconstriction with an overall decrease in TPR
Increased Heart Rate with a decrease in venous tone.
Increased Heart Rate with increased end systolic volume.
Review of Systems
Does not belong in patient write up as it is an opinion of the patient