Glands, heart, vessels
Excitation of the SA node
Closure of the heart valves
Friction of blood against the chamber walls
Opening and closing of the heart valves
Ventricles are in diastole
Blood enters pulmonary artieries and the aorta
Av valves are closed
Ventricles are in systole
A lowering of blood pressure due to change in cardiac output
A rise in blood pressure due to change in cardiac output
No change in blood pressure but a slower heart rate
No change in blood pressure but a change in respiration
Acoommodate a greater volume of blood
Expand the thracic cage during diastole
Pump blood with greater pressure
Pump blood through a smaller valve
SA node
AV valves
AV bundles
AV node
Ventricular repolarization
Ventricular depolarization
Atrial repolarization
Atrial depolarization
Right atrium
Left atrium
Right ventricle
Left ventricle
Trabeculae carneae
Pectinate muscles
Papillary muscles
Venae cavae
Ischemia
Pericarditis
Myocardial infarct
Angina pectoris
Second intercostal space to the right of the sternum
Second intercostal space to the left of the sternum
Fifth intercostal space inferior to the left nipple
Fifth right intercostal space
Coronary sinus
Fossa ovalis
Coronary arteries
Coronary veins
Pumps a greater volume of blood
Pumps blood against a greater resistance
Expands the thoracic cage
Sends blood through a smaller valve
Skin color
Age
Gender
Body temperature
Atherosclerosis
Decline in cardiac reserve
Fibrosis of cardiac muscle
Thinning of the valve flaps
Decreased delivery of oxygen
A decrease in the number of available mitochondria for energy production
A lack of nutrients to feed into metabolic pathways
An inadequate supply of lactic acid
It would be much longer before cardiac cells could respond to a second stimulation
Contractions would last as long as the refractory period
Tetanic contractions might occur, which would stop the hearts pumping action
It would be less than 1 2 ms
Decreasing heart contractility
Causing a decrease in stroke volume
Blocking the action of calcium
Causing threshold to be reached more quickly
The heart would stop, since the vagal nerves trigger the heart to contract
The heart rate would increase by about 25 beats per minute
The av node would become the pacemaker of the heart
Parasympathetic stimulation would increase, causing a decrease in heart rate
Connects the two atria in the fetal heart
Is a condition in which the heart valves do not completley close
Is a shallow depression in the intreventricular septum
Is a connection between the pulmonary trunk and the aorta in the fetus
Venae cavae
Pulmonary trunk
Aorta
Pulmonary veins
Aorta only
Pulmonary arteries only
Pulmonary veins only
Both aorta and pulmonary trunk
AV node
Bundle of His
AV valve
SA node
While the ventricle is in diastole
When the ventricle is in systole
While the atrium is contracting
By the movement of blood from the atrium to ventricle
Tracing out where the auricles connect
Noticing the thickness of the venricle walls
Locating the apex
Finding the papillary muscles
The mitral valve seperates the right atrium from the right ventricle
The tricuspid valve divides the left atrium from the left ventricle
Aortic and pulmonary valves control the flow of blood into the heart
The AV valves are supported by chordae tendineae so that regurgitation of blood into the atria during ventricle contaction does not occur
The entire heart contracts as a unit or it does not contract at all
Each cardiac muscle cell is nnervated by a sympathetic nerve ending so that the nervous system can increase heart rate
The refactory period in skeletal muscle is much longer than that in cardiac muscle
The influx of potassium ions from extracellular sources is the initiating event in cardiac muscle contraction.
The fibrous skeleton forms the bulk of the heart
Connective tissue in the heart wall aids in the conduction of the action potential
The heart chambers are lined by the endomysium
They myocardium is the layer of the heart that actually contracts
Has gap junctions that allow it to act as a functional syncytium
Lacks striations
Has more nuclei per cell
Cells are larger than skeletal muscle cells
Pressure in the heart is at its peak
Blood flows passively through the atria and the open AV valves into the ventricles
The atria remain in diastole
It is represented by the P wave on the ECG
Isovolumetric relaxation
Isovolumetric contraction
Ventricular ejection
Ventricular filling
Morning
Noon time
Evening
During sleep
Threshold is reached more quickly and heart rate would increase
Potassium channels compensate and no change in heart rate would occur
Heart rate would decrease, but blood pressure would rise due to the excess sodium present
Tetanic contraction would occur due to the short absolute refractory period of cardiac muscle
A slow heart rate increases end diastolic volume, stroke volume, and force of contraction
Decreased venous return will result in increased end diastolic volume
If a semilunar valve were partially obstructed, the end systolic volume in the affected ventricle would be decreased
Stroke volume increasees if end diastolic volume decreases
The action potential is initiated by voltage-gated slow calcium channels
Some calcium enters the cell from the extracellular space and triggers the release of larger amounts of calcium from intracellular stores
The action potential is prevented from spreading from cell to cell by gap junctions
Calcium is prevented from entering cardiac fibers that have been stimulated
Refers to the short period during ventricular systole when the ventricles are completely
Occurs while the AV valves are open
Occurs immediately after the aortic and pulmonary valves close
Occurs only in people with heart valve defects
Mild electrical shock to the heart itself
Severe electrical shock to the body
Relatively mild blow to the chest that occurs during a vulnerable interval (2 ms) when the heart is repolarizing
Loss of blood from an artery
Cardiac output
Peripheral resistance
Emotional state
Blood volume
ADH
Atrial natriuretic peptide
Angiotensin II
Nitric acid
All carry oxygenated blood to the heart
All carry blood away from the heart
All contain valves to prevent the backflow of blood
Only large arteries are lined with endothelium
Tunica intima
Tunica media
Tunica externa
Basement membrane
Arterioles
Arteries
Veins
Capillaries
Hepatic portal circulation
Pulmonary circulation
Coronary circulation
Cerebral circulation
Elastic arteries
Muscular arteries
Arterioles
Capillaries
Promote an increase in blood pressure
Promote a decrease in blood volume
Result in a larger ouput of urine
Decrease sodium reabsorption
Systolic pressure plus diastolic pressure
Systolic pressure minus diastolic pressure
Systolic pressure divided by diastolic pressure
Diastolic pressure plus 1/3 (systolic pressure plus diastolic pressure)
Cold, clammy skin
Increased heart rate
Rapid, thready pulse
Rapildy falling blood pressure
Blood will be deverted to the digestive organs
The skin will be cold and clammy
Capillaries of the active muscles will be engorged with blood
Blood flow to the kidneys increases
Their prime function is the exchange of nutrients and wastes between the blood and tissue cells
The contraction and realxation of the smooth muscle in their walls can change their diameter
They distribute blood to various parts of the body
They contain a large quantity of elastic tissue
Venous valves are formed from the tunica media
Up to 35% of total body blood is in venous circulation at any given time
Veins have a small lumen in relation to the thickness of the vessel wall
Veins are called capacitance vessels or blood reservoirs
Neural controls
Baroreceptor-initiated reflexes
Chemoreceptor-initiated reflexes
Renal regulation
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