Path exam 1
Necrosis
Apoptosis
Apoptosis
Necrosis
Apoptosis
Necrosis
Commitment
Control Stage
Execution stage
Phagocytosis
Commitment
Control Stage
Execution Stage
Phagocytosis
Death Receptor
P53
IL-2
TNF
P53
IL2
TNF
FasL
Caspase domain
Death Domain
BIR domains
Bcl-2 homology
P53
Bcl-2
Apaf-1
Procaspase 9
They are all members of the apoptosome
Bcl-2
Apaf-1
FasL
Il-2
None of the above
By dimerization via release of cytochrome C by the apoptosome
Through a singnaling cascade started by TNF
Through a ligand binding to a death domain
Through stimulation of BIR domains
BIR domains (IAPs)
Caspase associated recruitment domains
Bcl-2 homology domains
Caspase (catalytic) domains
None of the above
9
3
6
7
9
8
3
7
6
Protein Cross-Linking (activation of transglutaminase)
Internucleosomal cleavage of DNA into oligonucleosomes
Phagocytic Recognition Signals
None of the above
Cytokines
TNF
Fas ligand
TRAIL
None of the above
TNF to TNFR1
FasL to Fas
TRAIL to DR3
TRAIL to NF-kB
Glucocorticoid interaction with nuclear receptors
Hypoxia occurring in the mitochondria
Viral entrance to the cell
Clustering of intracellular, frequently nuclear receptors
Fas-FasL
Activation of TNF receptors
Cytotoxic T-Lymphocyte Stimulate apoptosis
Lack of integrin Signaling
None of the Above
Fas-FasL binding
Activation of TNF receptors
Cytotoxic T-Lymphocyte Stimulated apoptosis
Release of cytochrome C into the cytosol
Fas-FasL binding
TNF receptor association with protein TRADD
Cytotoxic T Lympocyte stimulated apoptosis
Integrin signaling apoptosis
Fas-FasL binding
TNF receptor association with adapter protein TRADD
Cytotoxic T- Lympocyte Simulated apoptosis
Integrin Signaling
BCL-2 family members Bcl-2 Bcl-XL, Mcl-1, A1
BCL-2 members Bad, Bak, Bax, Bcl-XS
Bax homodimers
Apaf-1
They all result in apoptosis
True
False
1
2
3
Rate this question:
1
2
3
Rate this question:
1
2
3
Rate this question:
8
9
3
7
8
9
3
10
3
7
2
12
9
8
3
1
1
3
7
8
Edema
Hyperemia
Hemorrhage
Hemostasis
True
False
More
Less
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