B. anabolic processes
C. catabolic processes
A. parietal cells
B. chief cells
C. mucous cells
D. gastrin cells
A. bicarbonate ion
B. hydrochloric acid
C. activated digestive enzymes
A. activate pepsinogen
B. activate intestinal and pancreatic enzymes
C. activate bile salts
D. produce mucus
A. They give feces the characteristic brown color.
B. They are enzymes used to break down fats into free fatty acids.
C. They emulsify lipids and lipid-soluble vitamins.
D. They are excreted in the feces.
A. metabolic alkalosis
B. metabolic acidosis
C. increased respirations
D. increased excretion of hydrogen ions
A. catabolism of proteins and lipids
B. continued loss of gastric secretions
C. loss of pancreatic enzymes
D. retention of sodium ions and water
A. increased bicarbonate ion
B. decreased bicarbonate ion
C. increased carbonic acid
D. increased serum pH
A. ketones produced
B. CO2 retained in the lungs and kidneys
C. hypovolemia and lactic acid production
D. metabolic rate
A. a large volume of chyme has entered the intestines, causing distention
B. severe hypoglycemia has developed
C. the pylorus is restricting the flow of chyme
D. bile and pancreatic secretions are irritating the small intestine
A. increased unconjugated bilirubin in the blood
B. increased bleeding tendencies
C. pale stool and dark urine
D. elevated liver enzymes in the blood
A. bilirubin or cholesterol concentration in the bile
B. water content in the bile
C. bile salts in the bile
D. bicarbonate ions in the bile
A. It is also called serum hepatitis.
B. It is transmitted by the fecal-oral route.
C. It contains a double strand of DNA.
D. It frequently leads to chronic hepatitis.
A. Acute infection is present.
B. Chronic infection has developed.
C. Liver failure is in progress.
D. The usual prolonged recovery from any viral infection is occurring.
A. systemic effects of viral infection
B. obstruction of bile ducts and malabsorption
C. necrosis of liver cells
D. ammonia toxicity
A. full recovery to normal tissue after the toxic material is removed
B. acute onset of vomiting, steatorrhea, and jaundice
C. continued mild inflammation of the liver without permanent damage
D. gradual irreversible damage to the liver and cirrhosis
A. below normal blood levels of AST and ALT
B. ULQ tenderness and dull pain
C. a small, firm, nodular liver and portal hypertension
D. accumulation of fat in the hepatocytes and hepatomegaly
A. serum urea
B. conjugated bilirubin
C. serum ammonia
D. serum pH
A. ingesting excessive lipids
B. bleeding occurs in the digestive tract
C. an increase in unconjugated bilirubin occurs in the serum
D. less bile is produced
A. portal hypertension
B. alcohol irritating the mucosa
C. failure to inactivate estrogen
D. poor nutritional status
A. anemia and leucopenia
B. jaundice and pruritus
C. recurrent infections
D. deficit of vitamin K and prothrombin